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Find Clinical Drug Pipeline Developments & Deals by Terns Pharmaceuticals
TERN-701 is company’s internally discovered allosteric BCR-ABL tyrosine kinase inhibitor, which is being evaluated for the treatment of chronic myeloid leukemia.
TERN-601 is an oral, small-molecule glucagon-like peptide-1 receptor, or GLP-1R, agonist program for obesity. Obesity is a chronic disease that is increasing in prevalence in adults, adolescents and children and is often defined by having an elevated BMI of 30 or greater.
TERN-701 is Terns’ proprietary, oral, potent, allosteric BCR-ABL TKI specifically targeting the BCR-ABL myristoyl pocket, which is in clinical development for chronic myeloid leukemia.
TERN-501 is a thyroid hormone receptor beta (THR-β) agonist with high metabolic stability, enhanced liver distribution and greater selectivity for THR-β compared to other THR-β agonists in development for the treatment of NASH.
TERN-501 is a Thyroid Hormone Receptor beta (THR-β) agonist with high metabolic stability, enhanced liver distribution and greater selectivity for THR-β compared to other THR-β agonists in development.
TERN-501 is a Thyroid Hormone Receptor beta (THR-β) agonist with high metabolic stability, enhanced liver distribution and greater selectivity for THR-beta compared to other THR-β agonists in development.
TERN-501 is a thyroid hormone receptor beta (THR-β) agonist with high metabolic stability, enhanced liver distribution and greater selectivity for THR-β compared to other THR-β agonists in development.
Oral presentation of data from Phase 1 first-in-human trial of TERN-501, demonstrating treatment was well-tolerated and resulted in significant dose-dependent effects on key target engagement biomarkers.
Multiple doses of thyroid hormone receptor-beta agonist TERN-501 were well-tolerated and resulted in significant dose-dependent changes in serum lipids and sex hormone binding globulin in a first-in-human clinical study.
Part 1 of AVIATION Trial met primary safety endpoint compared to placebo, for TERN-201, a highly selective, irreversible, covalent inhibitor of VAP-1, administered for 12 weeks to presumed NASH patients was generally well-tolerated with a similar incidence of adverse events.
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