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Chemistry

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Also known as: 29122-68-7, Tenormin, Blokium, Myocord, Normiten, Prenormine
Molecular Formula
C14H22N2O3
Molecular Weight
266.34  g/mol
InChI Key
METKIMKYRPQLGS-UHFFFAOYSA-N
FDA UNII
50VV3VW0TI
Atenolol
A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.
Atenolol is a beta-Adrenergic Blocker. The mechanism of action of atenolol is as an Adrenergic beta-Antagonist.
1 2D Structure

Atenolol

2 Identification
2.1 Computed Descriptors
2.1.1 IUPAC Name
2-[4-[2-hydroxy-3-(propan-2-ylamino)propoxy]phenyl]acetamide
2.1.2 InChI
InChI=1S/C14H22N2O3/c1-10(2)16-8-12(17)9-19-13-5-3-11(4-6-13)7-14(15)18/h3-6,10,12,16-17H,7-9H2,1-2H3,(H2,15,18)
2.1.3 InChI Key
METKIMKYRPQLGS-UHFFFAOYSA-N
2.1.4 Canonical SMILES
CC(C)NCC(COC1=CC=C(C=C1)CC(=O)N)O
2.2 Other Identifiers
2.2.1 UNII
50VV3VW0TI
2.3 Synonyms
2.3.1 MeSH Synonyms

1. Ici 66082

2. Ici-66082

3. Ici66082

4. Tenormin

5. Tenormine

2.3.2 Depositor-Supplied Synonyms

1. 29122-68-7

2. Tenormin

3. Blokium

4. Myocord

5. Normiten

6. Prenormine

7. Tenormine

8. (rs)-atenolol

9. Duraatenolol

10. Betacard

11. Corotenol

12. Tenoblock

13. Atehexal

14. Betablok

15. Cuxanorm

16. Juvental

17. Selobloc

18. Antipressan

19. Atcardil

20. Atenblock

21. Evitocor

22. Farnormin

23. Internolol

24. Normalol

25. Premorine

26. Prenolol

27. Tenoprin

28. Tensimin

29. Vascoten

30. Vericordin

31. Alinor

32. Anselol

33. Atecard

34. Atendol

35. Atenet

36. Atenil

37. Atereal

38. Aterol

39. Hipres

40. Hypoten

41. Ibinolo

42. Lotenal

43. Oraday

44. Serten

45. Stermin

46. Tenidon

47. Tenolol

48. Tredol

49. Uniloc

50. Wesipin

51. Altol

52. Ateni

53. Noten

54. Xaten

55. Seles Beta

56. Apo-atenolol

57. Felo-bits

58. Lo-ten

59. Atenolin

60. Atenomel

61. Blocotenol

62. Cardaxen

63. Cardiopress

64. Jenatenol

65. Panapres

66. Plenacor

67. Servitenol

68. Tenobloc

69. Aircrit

70. Betasyn

71. Ormidol

72. Prinorm

73. Unibloc

74. Loten

75. Atenol Acis

76. Atenol Cophar

77. Atenol Fecofar

78. Atenol Heumann

79. Atenol Nordic

80. Atenol Quesada

81. Atenol Gador

82. Atenol Stada

83. Atenol-mepha

84. Atenol-wolff

85. Atenol Atid

86. Atenol Ct

87. Atenol Tika

88. Atenol Trom

89. Atenol Genericon

90. Betatop Ge

91. Atenol Von Ct

92. Atenol-ratiopharm

93. Atenol Al

94. Atenol Pb

95. Atenol Gnr

96. Atenol Msd

97. Atenol Nm Pharma

98. Scheinpharm Atenol

99. Atenol 1a Pharma

100. Atenololum

101. 2-(4-(2-hydroxy-3-(isopropylamino)propoxy)phenyl)acetamide

102. Tenormine [french]

103. (r,s)-atenolol

104. Atenololum [inn-latin]

105. Ici 66082

106. 2-[4-[2-hydroxy-3-(propan-2-ylamino)propoxy]phenyl]acetamide

107. Tenormin (tn)

108. Ici-66082

109. Novaten

110. 1-p-carbamoylmethylphenoxy-3-isopropylamino-2-propanol

111. Ici 66,082

112. 2-[4-(2-hydroxy-3-isopropylaminopropoxy)phenyl]acetamide

113. 2-(p-(2-hydroxy-3-(isopropylamino)propoxy)phenyl)acetamide

114. 4-(2-hydroxy-3-((1-methylethyl)amino)propoxy)benzeneacetamide

115. 2-(4-{2-hydroxy-3-[(propan-2-yl)amino]propoxy}phenyl)acetamide

116. Benzeneacetamide, 4-[2-hydroxy-3-[(1-methylethyl)amino]propoxy]-

117. C07ab03

118. Chembl24

119. Duratenol

120. Nsc-757832

121. Benzeneacetamide, 4-(2-hydroxy-3-((1-methylethyl)amino)propoxy)-

122. 50vv3vw0ti

123. Mls000069622

124. Chebi:2904

125. Atenolol Bp

126. 60966-51-0

127. Smr000036768

128. Atenol

129. 2-{4-[2-hydroxy-3-(propan-2-ylamino)propoxy]phenyl}acetamide

130. Acetamide, 2-(p-(2-hydroxy-3-(isopropylamino)propoxy)phenyl)-

131. Teno-basan

132. Neatenol

133. Tensotin

134. Atcard

135. Dl-atenolol

136. Ccris 4196

137. (+/-)-atenolol

138. Hsdb 6526

139. Sr-01000000159

140. Einecs 249-451-7

141. Einecs 262-544-7

142. Mfcd00057645

143. Unii-50vv3vw0ti

144. Brn 2739235

145. 2-(4-[2-hydroxy-3-(isopropylamino)propoxy]phenyl)acetamide

146. Artrenolol

147. 2-(4-(2-hydroxy-3-(isopropylamino)propoxy)phenyl)ethanamide

148. 4-(2-hydroxy-3-[(1-methylethyl)amino]propoxy)benzeneacetamide

149. 2-(4-(2-hydroxy-3-isopropylaminopropoxy)phenyl)acetamid

150. Phenyl)acetamide

151. Atenolol [usan:ban:inn:jan]

152. (y)-atenolol

153. Atenalol (rs)

154. Atenolol,(s)

155. (?)-atenolol

156. Atenolol [usan:usp:inn:ban:jan]

157. (a+/-)-atenolol

158. Tenoretic (salt/mix)

159. Atenolol (jan/usp)

160. Spectrum_001364

161. Atenolol [hsdb]

162. Atenolol [usan]

163. Atenolol [inn]

164. Atenolol [jan]

165. Atenolol [mi]

166. Atenolol [vandf]

167. Opera_id_1283

168. Spectrum2_001411

169. Spectrum3_001448

170. Spectrum4_000435

171. Spectrum5_001509

172. Atenolol [mart.]

173. Dsstox_cid_2628

174. Atenolol [usp-rs]

175. Atenolol [who-dd]

176. Atenolol [who-ip]

177. A 7655

178. Schembl4362

179. Dsstox_rid_76663

180. Dsstox_gsid_22628

181. Lopac0_000121

182. Oprea1_448775

183. Bspbio_002915

184. Gtpl548

185. Kbiogr_000790

186. Kbioss_001844

187. Mls001066372

188. Mls001074163

189. Mls001304038

190. Divk1c_000057

191. Spectrum1501127

192. Atenolol (jp17/usp/inn)

193. Spbio_001482

194. Atenolol [orange Book]

195. Atenolol [ep Monograph]

196. Atenolol [usp Impurity]

197. Atenolol [usp Monograph]

198. Dtxsid2022628

199. Bdbm25753

200. Hms500c19

201. Kbio1_000057

202. Kbio2_001844

203. Kbio2_004412

204. Kbio2_006980

205. Kbio3_002415

206. Metkimkyrpqlgs-uhfffaoysa-

207. Polycap Component Atenolol

208. Atenololum [who-ip Latin]

209. Ninds_000057

210. S-atenolol-d7 (iso-propyl-d7)

211. 2-{4-[2-hydroxy-3-(isopropylamino)propoxy]-phenyl}acetamide

212. Hms1569l13

213. Hms1921h09

214. Hms2090i19

215. Hms2092d19

216. Hms2233e06

217. Hms3259k08

218. Hms3260i04

219. Hms3266k13

220. Hms3369b14

221. Hms3369d20

222. Hms3369p20

223. Hms3411g21

224. Hms3675g21

225. Hms3886g03

226. Pharmakon1600-01501127

227. ( Inverted Question Mark)-atenolol

228. Tenoretic Component Atenolol

229. Bcp12899

230. Atenolol 1.0 Mg/ml In Acetonitrile

231. Atenolol, >=98% (tlc), Powder

232. Tox21_302426

233. Tox21_500121

234. 2-[4-({2-hydroxy-3-[(1-methylethyl)amino]propyl}oxy)phenyl]acetamide

235. Atenolol 100 Microg/ml In Methanol

236. Bbl009276

237. Ccg-39010

238. Geo-03413

239. Nsc757832

240. S4817

241. Stk528649

242. Akos005111050

243. Atenolol Component Of Tenoretic

244. Ac-8245

245. Atenolol, Analytical Reference Material

246. Db00335

247. Ks-5341

248. Lp00121

249. Nc00548

250. Nsc 757832

251. Sdccgsbi-0050109.p004

252. Idi1_000057

253. Mrf-0000571

254. Ncgc00015007-06

255. Ncgc00015007-07

256. Ncgc00015007-08

257. Ncgc00015007-09

258. Ncgc00015007-10

259. Ncgc00015007-11

260. Ncgc00015007-13

261. Ncgc00015007-24

262. Ncgc00024566-03

263. Ncgc00024566-04

264. Ncgc00024566-05

265. Ncgc00024566-06

266. Ncgc00024566-07

267. Ncgc00255122-01

268. Ncgc00260806-01

269. Ba166036

270. Hy-17498

271. Sbi-0050109.p003

272. Cas-29122-68-7

273. Db-072177

274. Db-079552

275. Eu-0100121

276. Ft-0662315

277. Ft-0662316

278. Ft-0693045

279. 2-(4-(2-hydroxy-3-(isopropylamino)propoxy)

280. Bim-0050109.0001

281. D00235

282. O10469

283. Ab00052208-13

284. Ab00052208-15

285. Ab00052208_16

286. 122a687

287. L000116

288. Q411325

289. Q-200656

290. Sr-01000000159-2

291. Sr-01000000159-4

292. Sr-01000000159-5

293. Sr-01000000159-8

294. Brd-a20239487-001-02-5

295. Brd-a20239487-001-15-7

296. Atenolol, European Pharmacopoeia (ep) Reference Standard

297. Z1541638518

298. Atenolol, United States Pharmacopeia (usp) Reference Standard

299. (+)-4-[2-hydroxy-3-[(1-methylethyl)amino]propoxy]benzeneacetamide

300. (rs)-4-[2-hydroxy-3-[(1-methylethyl)amino]propoxy]benzeneacetamide

301. 2-(p-(hydroxy-3-(isopropylamino)propoxy)phenyl)acetamide

302. (+/-)-4-(2-hydroxy-3-[(1-methylethyl)amino]propoxy)benzeneacetamide

303. 2-(4-{[(2s)-2-hydroxy-3-(propan-2-ylamino)propyl]oxy}phenyl)acetamide

304. Atenolol, Pharmaceutical Secondary Standard; Certified Reference Material

305. 2-(p-(2-hydroxy-3-(isopropylamino)propoxy)phenyl)acetamide (racemate)

306. 2-[4-({(2r)-2-hydroxy-3-[(1-methylethyl)amino]propyl}oxy)phenyl]acetamide

307. Atenolol Solution, 1.0 Mg/ml In Acetonitrile, Ampule Of 1 Ml, Certified Reference Material

308. 106020-65-9

2.4 Create Date
2005-03-25
3 Chemical and Physical Properties
Molecular Weight 266.34 g/mol
Molecular Formula C14H22N2O3
XLogP30.2
Hydrogen Bond Donor Count3
Hydrogen Bond Acceptor Count4
Rotatable Bond Count8
Exact Mass266.16304257 g/mol
Monoisotopic Mass266.16304257 g/mol
Topological Polar Surface Area84.6 Ų
Heavy Atom Count19
Formal Charge0
Complexity263
Isotope Atom Count0
Defined Atom Stereocenter Count0
Undefined Atom Stereocenter Count1
Defined Bond Stereocenter Count0
Undefined Bond Stereocenter Count0
Covalently Bonded Unit Count1
4 Drug and Medication Information
4.1 Drug Information
1 of 4  
Drug NameAtenolol
PubMed HealthAtenolol
Drug ClassesAntianginal, Antiarrhythmic, Group II, Antihypertensive, Antimigraine, Cardiovascular Agent
Drug LabelAtenolol, a synthetic, beta1-selective (cardioselective) adrenoreceptor blocking agent, may be chemically described as benzeneacetamide, 4 -[2'-hydroxy-3'-[(1- methylethyl) amino] propoxy]-. The molecular formula is C14H22N2O3 and its structural
Active IngredientAtenolol
Dosage FormTablet
RouteOral
Strength25mg; 100mg; 50mg
Market StatusPrescription
CompanyIpca Labs; Mutual Pharm; Teva; Ipr; Aurobindo Pharma; Sun Pharm Inds; Zydus Pharms Usa; Northstar Hlthcare; Unique Pharm Labs; Sandoz; Mylan; Dava Pharms

2 of 4  
Drug NameTenormin
PubMed HealthAtenolol
Drug ClassesAntianginal, Antiarrhythmic, Group II, Antihypertensive, Antimigraine, Cardiovascular Agent
Drug LabelTENORMIN (atenolol), a synthetic, beta1-selective (cardioselective) adrenoreceptor blocking agent, may be chemically described as benzeneacetamide, 4 -[2'-hydroxy-3'-[(1- methylethyl) amino] propoxy]-. The molecular and structural formulas are:
Active IngredientAtenolol
Dosage FormTablet
RouteOral
Strength25mg; 100mg; 50mg
Market StatusPrescription
CompanyAstrazeneca

3 of 4  
Drug NameAtenolol
PubMed HealthAtenolol
Drug ClassesAntianginal, Antiarrhythmic, Group II, Antihypertensive, Antimigraine, Cardiovascular Agent
Drug LabelAtenolol, a synthetic, beta1-selective (cardioselective) adrenoreceptor blocking agent, may be chemically described as benzeneacetamide, 4 -[2'-hydroxy-3'-[(1- methylethyl) amino] propoxy]-. The molecular formula is C14H22N2O3 and its structural
Active IngredientAtenolol
Dosage FormTablet
RouteOral
Strength25mg; 100mg; 50mg
Market StatusPrescription
CompanyIpca Labs; Mutual Pharm; Teva; Ipr; Aurobindo Pharma; Sun Pharm Inds; Zydus Pharms Usa; Northstar Hlthcare; Unique Pharm Labs; Sandoz; Mylan; Dava Pharms

4 of 4  
Drug NameTenormin
PubMed HealthAtenolol
Drug ClassesAntianginal, Antiarrhythmic, Group II, Antihypertensive, Antimigraine, Cardiovascular Agent
Drug LabelTENORMIN (atenolol), a synthetic, beta1-selective (cardioselective) adrenoreceptor blocking agent, may be chemically described as benzeneacetamide, 4 -[2'-hydroxy-3'-[(1- methylethyl) amino] propoxy]-. The molecular and structural formulas are:
Active IngredientAtenolol
Dosage FormTablet
RouteOral
Strength25mg; 100mg; 50mg
Market StatusPrescription
CompanyAstrazeneca

4.2 Therapeutic Uses

Adrenergic beta-Antagonists; Anti-Arrhythmia Agents; Antihypertensive Agents; Sympatholytics

National Library of Medicine's Medical Subject Headings online file (MeSH, 1999)

Atenolol has been used with good results alone or in conjunction with a benzodiazepine in the management of acute alcohol withdrawal in a limited number of patients.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1739

Atenolol ... /is/ indicated in the treatment of classic angina pectoris, also referred to as "effort-associated angina". /Included in US product labeling/

MICROMEDEX Thomson Health Care. USPDI - Drug Information for the Health Care Professional. 23rd ed. Volume 1. MICROMEDEX Thomson Health Care, Greenwood Village, CO. 2003. Content Reviewed and Approved by the U.S. Pharmacopeial Convention, Inc., p. 550

Atenolol /is/ used in the treatment of mitral value prolapse syndrome. /NOT included in US product labeling/

MICROMEDEX Thomson Health Care. USPDI - Drug Information for the Health Care Professional. 23rd ed. Volume 1. MICROMEDEX Thomson Health Care, Greenwood Village, CO. 2003. Content Reviewed and Approved by the U.S. Pharmacopeial Convention, Inc., p. 551

For more Therapeutic Uses (Complete) data for ATENOLOL (12 total), please visit the HSDB record page.

4.3 Drug Warning

Atenolol should be used with caution and in reduced dosage in patients with impaired renal function, especially when creatinine clearance is less than 35 ml/minute per 1.73 sq m. ... Patients receiving atenolol after hemodialysis /should/ be administered the drug under close supervision in a hospital setting, since marked hypotension may occur.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1742

Atenolol is contraindicated in patients with sinus bradycardia, AV block greater than first degree, cardiogenic shock, and overt cardiac failure.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1742

Atenolol should be used with caution in patients undergoing major surgery involving general anesthesia. The necessity of withdrawing beta-adrenergic blocking therapy prior to major surgery is controversial. Severe, protracted hypotension and difficulty in restarting or maintaining a heart beat have occurred during surgery in some patients who have received beta-adrenergic blocking agents.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1742

Abrupt withdrawal of atenolol may exacerbate angina symptoms and/or precipitate myocardial infarction and venticular arrhythmias in patients with coronary artery disease, or may precipitate thyroid storm in patients with thyrotoxicosis. Therefore, patients receiving atenolol (especially those with ischemic heart disease) should be warned not to interrupt or discontinue therapy without consulting their physician.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1741

For more Drug Warnings (Complete) data for ATENOLOL (12 total), please visit the HSDB record page.

4.4 Drug Indication

**Indicated** for: 1) Management of hypertension alone and in combination with other antihypertensives. 2) Management of angina pectoris associated with coronary atherosclerosis. 3) Management of acute myocardial infarction in hemodynamically stable patients with a heart rate greater than 50 beats per minutes and a systolic blood pressure above 100 mmHg. **Off-label** uses include: 1) Secondary prevention of myocardial infarction. 2) Management of heart failure. 3) Management of atrial fibrillation. 4) Management of supraventricular tachycardia. 5) Management of ventricular arrythmias such as congenital long-QT and arrhythmogenic right ventricular cardiomyopathy. 6) Management of symptomatic thyrotoxicosis in combination with [methimazole]. 7) Prophylaxis of migraine headaches. 8) Management of alcohol withdrawal.

FDA Label

5 Pharmacology and Biochemistry
5.1 Pharmacology

Atenolol is a cardio-selective beta-blocker and as such exerts most of its effects on the heart. It acts as an antagonist to sympathetic innervation and prevents increases in heart rate, electrical conductivity, and contractility in the heart due to increased release of norepinephrine from the peripheral nervous system. Together the decreases in contractility and rate produce a reduction in cardiac output resulting in a compensatory increase in peripheral vascular resistance in the short-term. This response later declines to baseline with long-term use of atenolol. More importantly, this reduction in the work demanded of the myocardium also reduces oxygen demand which provides therapeutic benefit by reducing the mismatch of oxygen supply and demand in settings where coronary blood flow is limited, such as in coronary atherosclerosis. Reducing oxygen demand, particularly due to exercise, can reduce the frequency of angina pectoris symptoms and potentially improve survival of the remaining myocardium after myocardial infarction. The decrease in rate of sinoatrial node potentials, electrical conduction, slowing of potentials traveling through the atrioventricular node, and reduced frequency of ectopic potentials due to blockade of adrenergic beta receptors has led to benefit in arrhythmic conditions such as atrial fibrillation by controlling the rate of action potential generation and allowing for more effective coordinated contractions. Since a degree of sympathetic activity is necessary to maintain cardiac function, the reduced contractility induced by atenolol may precipitate or worsen heart failure, especially during volume overload. The effects of atenolol on blood pressure have been established, although it is less effective than alternative beta-blockers, but the mechanism has not yet been characterized. As a 1 selective drug, it does not act via the vasodilation produced by non-selective agents. Despite this there is a sustained reduction in peripheral vascular resistance, and consequently blood pressure, alongside a decrease in cardiac output. It is thought that atenolol's antihypertensive activity may be related to action on the central nervous system (CNS) or it's inhibition of the renin-aldosterone-angiotensin system rather than direct effects on the vasculature. Atenolol produces CNS effects similar to other beta-blockers, but does so to a lesser extent due to reduces ability to cross the blood-brain barrier. It has the potential to produce fatigue, depression, and sleep disturbances such as nightmares or insomnia. The exact mechanisms behind these have not been characterized but their occurrence must be considered as they represent clinically relevant adverse effects. Atenolol exerts some effects on the respiratory system although to a much lesser extent than non-selective beta-blockers. Interaction with 2 receptors in the airways can produce bronchoconstriction by blocking the relaxation of bronchial smooth muscle mediated by the sympathetic nervous system. The same action can interfere with -agonist therapies used in asthma and chronic obstructive pulmonary disease. Unlike some other beta-blocker drugs, atenolol does not have intrinsic sympathomimetic or membrane stabilizing activity nor does it produce changes in glycemic control.

5.2 MeSH Pharmacological Classification

Anti-Arrhythmia Agents

Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade. (See all compounds classified as Anti-Arrhythmia Agents.)

Sympatholytics

Drugs that inhibit the actions of the sympathetic nervous system by any mechanism. The most common of these are the ADRENERGIC ANTAGONISTS and drugs that deplete norepinephrine or reduce the release of transmitters from adrenergic postganglionic terminals (see ADRENERGIC AGENTS). Drugs that act in the central nervous system to reduce sympathetic activity (e.g., centrally acting alpha-2 adrenergic agonists, see ADRENERGIC ALPHA-AGONISTS) are included here. (See all compounds classified as Sympatholytics.)

Adrenergic beta-1 Receptor Antagonists

Drugs that bind to and block the activation of ADRENERGIC BETA-1 RECEPTORS. (See all compounds classified as Adrenergic beta-1 Receptor Antagonists.)

Antihypertensive Agents

Drugs used in the treatment of acute or chronic vascular HYPERTENSION regardless of pharmacological mechanism. Among the antihypertensive agents are DIURETICS; (especially DIURETICS, THIAZIDE); ADRENERGIC BETA-ANTAGONISTS; ADRENERGIC ALPHA-ANTAGONISTS; ANGIOTENSIN-CONVERTING ENZYME INHIBITORS; CALCIUM CHANNEL BLOCKERS; GANGLIONIC BLOCKERS; and VASODILATOR AGENTS. (See all compounds classified as Antihypertensive Agents.)

5.3 FDA Pharmacological Classification
5.3.1 Active Moiety
ATENOLOL
5.3.2 FDA UNII
50VV3VW0TI
5.3.3 Pharmacological Classes
beta-Adrenergic Blocker [EPC]; Adrenergic beta-Antagonists [MoA]
5.4 ATC Code

C07AB03

S76 | LUXPHARMA | Pharmaceuticals Marketed in Luxembourg | Pharmaceuticals marketed in Luxembourg, as published by d'Gesondheetskeess (CNS, la caisse nationale de sante, www.cns.lu), mapped by name to structures using CompTox by R. Singh et al. (in prep.). List downloaded from https://cns.public.lu/en/legislations/textes-coordonnes/liste-med-comm.html. Dataset DOI:10.5281/zenodo.4587355

C07AB03

S66 | EAWAGTPS | Parent-Transformation Product Pairs from Eawag | DOI:10.5281/zenodo.3754448

C07AB03

S66 | EAWAGTPS | Parent-Transformation Product Pairs from Eawag | DOI:10.5281/zenodo.3754448

C - Cardiovascular system

C07 - Beta blocking agents

C07A - Beta blocking agents

C07AB - Beta blocking agents, selective

C07AB03 - Atenolol

5.5 Absorption, Distribution and Excretion

Absorption

Approximately 50% of an oral dose is absorbed from the gastrointestinal tract, with the remainder being excreted unchanged in the feces. Administering atenolol with food can decrease the AUC by about 20%. While atenolol can cross the blood-brain barrier, it does so slowly and to a small extent.

Route of Elimination

85% is eliminated by the kidneys following IV administration with 10% appearing in the feces.

Volume of Distribution

Total Vd of 63.8-112.5 L. Atenolol distributes into a central volume of 12.8-17.5 L along with two peripheral compartments with a combined volume of 51-95 L. Distribution takes about 3 hrs for the central compartment, 4 hrs for the shallower peripheral compartment, and 5-6 hrs for the deeper peripheral compartment.

Clearance

Total clearance is estimated at 97.3-176.3 mL/min with a renal clearance of 95-168 mL/min.

In animals, atenolol is well distributed into most tissues and fluids except brain and /cerebrospinal fluid/. Unlike propranolol, only a small portion of atenolol is apparently distributed into the CNS.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

Approximately 5-15% of atenolol is bound to plasma protein.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

Atenolol readily crosses the placenta, and has been detected in cord blood. During continuous administration, fetal serum concentrations of the drug are probably equivalent to those in maternal serum. Atenolol is distributed into milk; peak milk concentrations of the drug are higher than peak serum concentrations after an individual dose, and the area under the milk concentration-time (AUC) is substantially greater than that of the serum AUC in lactating women receiving the drug continuously.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

Atenolol is rapidly but incompletely absorbed from the GI tract. Only about 50-60% of an oral dose of atenolol is absorbed. In healthy adults, peak plasma concentrations of 1-2 ug/ml are achieved 2-4 hours after oral administration of a single 200 mg dose of atenolol. An approximately fourfold interindividual variation in plasma concentrations attained has been reported with a specific oral dose of atenolol. Peak plasma atenolol concentrations are achieved within 5 minutes following direct IV injection of the drug, and decline rapidly during an initial distribution phase; after the first 7 hours, plasma concentrations reportedly decline with an elimination half-life similar to that of orally administered drug.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

For more Absorption, Distribution and Excretion (Complete) data for ATENOLOL (6 total), please visit the HSDB record page.

5.6 Metabolism/Metabolites

Minimal metabolism in the liver. The sole non-conjugated metabolite is the product of a hydroxylation reaction at the carbon between the amide and benzene groups. The only other metabolite to be confirmed is a glucuronide conjugate. These metabolites make up 5-8% and 2% of the renally excreted dose with 87-90% appearing as unchanged drug. The hydroxylated metabolite is exerts 1/10th the beta-blocking activity of atenolol.

Minimal hepatic metabolism; removable by hemodialysis; very low lipid solubility.

US Pharmacopeial Convention; US Pharmacopeia Dispensing Information (USP DI); Drug Information for the Health Care Professional 12th ed, V.I p.635 (1992)

Little or no metabolism of atenolol occurs in the liver. Approximately 40-50% of an oral dose of the drug is excreted in urine unchanged. The remainder is excreted unchanged in feces, principally as unabsorbed drug. About 1-12% of atenolol is reportedly removed by hemodialysis.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

5.7 Biological Half-Life

6-7 hrs.

In patients with normal renal function, atenolol has a plasma half-life (t1/2) of 6-7 hours. Children with normal renal function may exhibit a shorter elimination half-life. In one study in children ages 5-16 (mean: 8.9) with arhythmias and normal renal and hepatic function, the terminal elimination half-life averaged 4.6 hours. Plasma t1/2 of the drug increases to 16-27 hours in patients with creatinine clearances of 15-35 ml/minute per 1.73 sq m and exceeds 27 hours with progressive renal impairment.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

The half-life in the elderly was significantly longer (8.8 + or - 0.9 hr) compared with that in the young (5.8 + or - 1.1 hr) (p < 0.01).

PMID:3358894 Full text: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1386352 Scott M et al; Br J Clin Pharmacol 25 (3): 289-96 (1988)

5.8 Mechanism of Action

Atenolol is a cardioselective beta-blocker, called such because it selectively binds to the 1-adrenergic receptor as an antagonist up to a reported 26 fold more than 2 receptors. Selective activity at the 1 receptor produces cardioselectivity due to the higher population of this receptor in cardiac tissue. Some binding to 2 and possibly 3 receptors can still occur at therapeutic dosages but the effects mediated by antagonizing these are significantly reduced from those of non-selective agents. 1 and 2 receptors are Gs coupled therefore antagonism of their activation reduces activity of adenylyl cyclase and its downstream signalling via cyclic adenosime monophosphate and protein kinase A (PKA). In cardiomyocytes PKA is thought to mediate activation of L-type calcium channels and ryanodine receptors through their phosphorylation. L-type calcium channels can then provide an initial rise in intracellular calcium and trigger the ryanodine receptors to release calcium stored in the sarcoplasmic reticulum (SR) and increased contractility. PKA also plays a role in the cessation of contraction by phosphorylating phospholamban which in turn increases the affinity of SR Ca2+ ATPase to increase reuptake of calcium into the SR. It also phophorylates troponin I to reduce affinity of the protein for calcium. Both of these events lead to a reduction in contraction which, when coupled with the initial increase in contraction, allows for faster cycling and consequently higher heart rate with increased contractility. L-type calcium channels are also a major contributor to cardiac depolarization and their activation can increase frequency of action potentials and possibly the incidence of ectopic potentials. Similar inihibitory events occur in the bronchial smooth muscle to mediate relaxation including phosphorylation of myosin light-chain kinase, reducing its affinity for calcium. PKA also inhibits the excitatory Gq coupled pathway by phosphorylating the inositol trisphosphate receptor and phospholipase C resulting in inhibition of intracellular calcium release. Antagonism of this activity by beta-blocker agents like atenolol can thus cause increased bronchoconstriction.

By inhibiting myocardial beta 1-adrenergic receptors, atenolol produces negative chronotropic and inotropic activity. The negative chronotropic action of atenolol on the sinoatrial node results in a decrease in the rate of sinoatrial node discharge and an increase in recovery time, thereby decreasing resting and exercise stimulated heart rate and reflex orthostatic tachycardia by about 25-35%. High doses of the drug may produce sinus arrest, especially in patients with sinoatrial node disease (eg, sick sinus syndrome). Atenolol also slows conduction in the atrioventricular nose. Although stroke index may be increased moderately by about 10%, atenolol usually reduces cardiac output by about 20% probably secondary to its effect on heart rate. The decrease in myocardial contractability and heart rate, as well as the reduction in blood pressure, produced by atenolol generally lead to a reduction in myocardial oxygen consumption which accounts for the effectiveness of the drug in chronic stable angina pectoris; however, atenolol can increase oxygen requirements by increasing left ventricular fiber length and end-diastolic pressure, particularly in patients with cardiac failure.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

Atenolol suppresses plasma renin activity and suppresses the renin aldosterone angiotensin system.

McEvoy, G.K. (ed.). American Hospital Formulary Service - Drug Information 2003. Bethesda, MD: American Society of Health-System Pharmacists, Inc. 2003 (Plus Supplements)., p. 1743

The toxic actions of beta-blockers appear to be related to properties such as membrane depressant activity and possibly due to actions on beta-adrenoceptors distinct from those in the cardiovascular system.

PMID:2565523 Critchley JA, Ungar A; Med Toxicol Adverse Drug Exp 4 (1): 32-45 (1989)

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BOX R1462, ROYAL EXCHANGE POST OFFICE SYDNEY NSW 1225 Sydney - ., , Australia Australia","customerAddress":"48, KANDIVLI INDUSTRIAL ESTATE,"}]
03-Jan-2022
25-Feb-2026
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DOSAGE - TABLET;ORAL - 100MG

USFDA APPLICATION NUMBER - 18240

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DOSAGE - TABLET;ORAL - 25MG

USFDA APPLICATION NUMBER - 18240

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DOSAGE - TABLET;ORAL - 50MG

USFDA APPLICATION NUMBER - 18240

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DOSAGE - TABLET;ORAL - 100MG;25MG

USFDA APPLICATION NUMBER - 18760

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DOSAGE - TABLET;ORAL - 50MG;25MG

USFDA APPLICATION NUMBER - 18760

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ABOUT THIS PAGE

Looking for 29122-68-7 / Atenolol API manufacturers, exporters & distributors?

Atenolol manufacturers, exporters & distributors 1

20

PharmaCompass offers a list of Atenolol API manufacturers, exporters & distributors, which can be sorted by GMP, USDMF, JDMF, KDMF, CEP (COS), WC, Price,and more, enabling you to easily find the right Atenolol manufacturer or Atenolol supplier for your needs.

Send us enquiries for free, and we will assist you in establishing a direct connection with your preferred Atenolol manufacturer or Atenolol supplier.

PharmaCompass also assists you with knowing the Atenolol API Price utilized in the formulation of products. Atenolol API Price is not always fixed or binding as the Atenolol Price is obtained through a variety of data sources. The Atenolol Price can also vary due to multiple factors, including market conditions, regulatory modifications, or negotiated pricing deals.

API | Excipient name

Atenolol

Synonyms

29122-68-7, Tenormin, Blokium, Myocord, Normiten, Prenormine

Cas Number

29122-68-7

Unique Ingredient Identifier (UNII)

50VV3VW0TI

About Atenolol

A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.

Tenormin Manufacturers

A Tenormin manufacturer is defined as any person or entity involved in the manufacture, preparation, processing, compounding or propagation of Tenormin, including repackagers and relabelers. The FDA regulates Tenormin manufacturers to ensure that their products comply with relevant laws and regulations and are safe and effective to use. Tenormin API Manufacturers are required to adhere to Good Manufacturing Practices (GMP) to ensure that their products are consistently manufactured to meet established quality criteria.

click here to find a list of Tenormin manufacturers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PhamaCompass.

Tenormin Suppliers

A Tenormin supplier is an individual or a company that provides Tenormin active pharmaceutical ingredient (API) or Tenormin finished formulations upon request. The Tenormin suppliers may include Tenormin API manufacturers, exporters, distributors and traders.

click here to find a list of Tenormin suppliers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PharmaCompass.

Tenormin USDMF

A Tenormin DMF (Drug Master File) is a document detailing the whole manufacturing process of Tenormin active pharmaceutical ingredient (API) in detail. Different forms of Tenormin DMFs exist exist since differing nations have different regulations, such as Tenormin USDMF, ASMF (EDMF), JDMF, CDMF, etc.

A Tenormin DMF submitted to regulatory agencies in the US is known as a USDMF. Tenormin USDMF includes data on Tenormin's chemical properties, information on the facilities and procedures used, and details about packaging and storage. The Tenormin USDMF is kept confidential to protect the manufacturer’s intellectual property.

click here to find a list of Tenormin suppliers with USDMF on PharmaCompass.

Tenormin JDMF

The Pharmaceuticals and Medical Devices Agency (PMDA) established the Japan Drug Master File (JDMF), also known as the Master File (MF), to permit Japanese and foreign manufacturers of drug substances, intermediates, excipients, raw materials, and packaging materials (‘Products’) to voluntarily register confidential information about the production and management of their products in Japan.

The Tenormin Drug Master File in Japan (Tenormin JDMF) empowers Tenormin API manufacturers to present comprehensive information (e.g., production methods, data, etc.) to the review authority, i.e., PMDA (Pharmaceuticals & Medical Devices Agency).

PMDA reviews the Tenormin JDMF during the approval evaluation for pharmaceutical products. At the time of Tenormin JDMF registration, PMDA checks if the format is accurate, if the necessary items have been included (application), and if data has been attached.

click here to find a list of Tenormin suppliers with JDMF on PharmaCompass.

Tenormin KDMF

In Korea, the Ministry of Food and Drug Safety (MFDS) is in charge of regulating pharmaceutical products and services.

Pharmaceutical companies submit a Tenormin Drug Master File in Korea (Tenormin KDMF) to the MFDS, which includes comprehensive information about the production, processing, facilities, materials, packaging, and testing of Tenormin. The MFDS reviews the Tenormin KDMF as part of the drug registration process and uses the information provided in the Tenormin KDMF to evaluate the safety and efficacy of the drug.

After submitting a Tenormin KDMF to the MFDS, the registered manufacturer can provide importers or distributors with the registration number without revealing confidential information to Korean business partners. Applicants seeking to register their Tenormin API can apply through the Korea Drug Master File (KDMF).

click here to find a list of Tenormin suppliers with KDMF on PharmaCompass.

Tenormin CEP

A Tenormin CEP of the European Pharmacopoeia monograph is often referred to as a Tenormin Certificate of Suitability (COS). The purpose of a Tenormin CEP is to show that the European Pharmacopoeia monograph adequately controls the purity of Tenormin EP produced by a given manufacturer. Suppliers of raw materials can prove the suitability of Tenormin to their clients by showing that a Tenormin CEP has been issued for it. The manufacturer submits a Tenormin CEP (COS) as part of the market authorization procedure, and it takes on the role of a Tenormin CEP holder for the record. Additionally, the data presented in the Tenormin CEP (COS) is managed confidentially and offers a centralized system acknowledged by numerous nations, exactly like the Tenormin DMF.

A Tenormin CEP (COS) is recognised by all 36 nations that make up the European Pharmacopoeia Convention. Tenormin CEPs may be accepted in nations that are not members of the Ph. Eur. at the discretion of the authorities there.

click here to find a list of Tenormin suppliers with CEP (COS) on PharmaCompass.

Tenormin WC

A Tenormin written confirmation (Tenormin WC) is an official document issued by a regulatory agency to a Tenormin manufacturer, verifying that the manufacturing facility of a Tenormin active pharmaceutical ingredient (API) adheres to the Good Manufacturing Practices (GMP) regulations of the importing country. When exporting Tenormin APIs or Tenormin finished pharmaceutical products to another nation, regulatory agencies frequently require a Tenormin WC (written confirmation) as part of the regulatory process.

click here to find a list of Tenormin suppliers with Written Confirmation (WC) on PharmaCompass.

Tenormin NDC

National Drug Code is a comprehensive database maintained by the FDA that contains information on all drugs marketed in the US. This directory includes information about finished drug products, unfinished drug products, and compounded drug products, including those containing Tenormin as an active pharmaceutical ingredient (API).

Finished drug products

The FDA updates the NDC directory daily. The NDC numbers for Tenormin API and other APIs are published in this directory by the FDA.

Unfinished drugs

The NDC unfinished drugs database includes product listing information submitted for all unfinished drugs, such as active pharmaceutical ingredients (APIs), drugs intended for further processing and bulk drug substances for compounding.

Pharmaceutical companies that manufacture Tenormin as an active pharmaceutical ingredient (API) must furnish the FDA with an updated record of all drugs that they produce, prepare, propagate, compound, or process for commercial distribution in the US at their facilities.

Compounded drug products

The NDC directory also contains data on finished compounded human drug products that contain Tenormin and are produced by outsourcing facilities. While these outsourcing facilities are not mandated to assign a Tenormin NDC to their finished compounded human drug products, they may choose to do so.

click here to find a list of Tenormin suppliers with NDC on PharmaCompass.

Tenormin GMP

Tenormin Active pharmaceutical ingredient (API) is produced in GMP-certified manufacturing facility.

GMP stands for Good Manufacturing Practices, which is a system used in the pharmaceutical industry to make sure that goods are regularly produced and monitored in accordance with quality standards. The FDA’s current Good Manufacturing Practices requirements are referred to as cGMP or current GMP which indicates that the company follows the most recent GMP specifications. The World Health Organization (WHO) has its own set of GMP guidelines, called the WHO GMP. Different countries can also set their own guidelines for GMP like China (Chinese GMP) or the EU (EU GMP).

PharmaCompass offers a list of Tenormin GMP manufacturers, exporters & distributors, which can be sorted by USDMF, JDMF, KDMF, CEP (COS), WC, API price, and more, enabling you to easily find the right Tenormin GMP manufacturer or Tenormin GMP API supplier for your needs.

Tenormin CoA

A Tenormin CoA (Certificate of Analysis) is a formal document that attests to Tenormin's compliance with Tenormin specifications and serves as a tool for batch-level quality control.

Tenormin CoA mostly includes findings from lab analyses of a specific batch. For each Tenormin CoA document that a company creates, the USFDA specifies specific requirements, such as supplier information, material identification, transportation data, evidence of conformity and signature data.

Tenormin may be tested according to a variety of international standards, such as European Pharmacopoeia (Tenormin EP), Tenormin JP (Japanese Pharmacopeia) and the US Pharmacopoeia (Tenormin USP).

Inform the supplier about your product requirements, specifying if you need a product with particular monograph like EP (Ph. Eur.), USP, JP, BP, or any other quality. In addition, clarify whether you need hydrochloride (HCl), anhydricum, base, micronisatum or a specific level of purity. To find reputable suppliers, utilize the filters and select those certified by GMP, FDA, or any other certification as per your requirement.
For your convenience, we have listed synonyms and CAS numbers to help you find the best supplier. The use of synonyms and CAS numbers can be helpful in identifying potential suppliers, but it is crucial to note that they might not always indicate the exact same product. It is important to confirm the product details with the supplier before making a purchase to ensure that it meets your requirements.
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