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    Chemistry

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    Also known as: 7782-44-7, O, Molecular oxygen, Oxygen molecule, Dioxygen, Pure oxygen
    Molecular Formula
    O2
    Molecular Weight
    31.999  g/mol
    InChI Key
    MYMOFIZGZYHOMD-UHFFFAOYSA-N
    FDA UNII
    S88TT14065
    Oxygen
    An element with atomic symbol O, atomic number 8, and atomic weight [15.99903; 15.99977]. It is the most abundant element on earth and essential for respiration.
    1 2D Structure

    Oxygen

    2 Identification
    2.1 Computed Descriptors
    2.1.1 IUPAC Name
    molecular oxygen
    2.1.2 InChI
    InChI=1S/O2/c1-2
    2.1.3 InChI Key
    MYMOFIZGZYHOMD-UHFFFAOYSA-N
    2.1.4 Canonical SMILES
    O=O
    2.2 Other Identifiers
    2.2.1 UNII
    S88TT14065
    2.3 Synonyms
    2.3.1 MeSH Synonyms

    1. Dioxygen

    2. Oxygen 16

    3. Oxygen-16

    2.3.2 Depositor-Supplied Synonyms

    1. 7782-44-7

    2. O

    3. Molecular Oxygen

    4. Oxygen Molecule

    5. Dioxygen

    6. Pure Oxygen

    7. Oxygenium

    8. Liquid Oxygen

    9. Hyperoxia

    10. Sauerstoff

    11. Oxygen-16

    12. Oxygen (liquid)

    13. Oxygen, Liquified

    14. Oxygenium Medicinale

    15. Oxygene

    16. Ccris 1228

    17. Hsdb 5054

    18. O2

    19. Un1072

    20. Un1073

    21. Rns60 Component Oxygen

    22. Ins No.948

    23. Peroxide Ion

    24. E-948

    25. Rns-60 Component Oxygen

    26. Chebi:15379

    27. Ins-948

    28. S88tt14065

    29. Lox

    30. E948

    31. Oxygene [french]

    32. Oxigeno [spanish]

    33. Oxy

    34. Compressed Oxygen

    35. Oxygen [usp]

    36. Einecs 231-956-9

    37. Dioxidanediyl

    38. Dioxygene

    39. Disauerstoff

    40. Oxygen Monoxide

    41. Peroxy Radical

    42. Singlet Dioxygen

    43. Triplet Dioxygen

    44. Unii-s88tt14065

    45. Oxygen Usp

    46. Oxygen, Compressed

    47. Oxygen 93 Percent

    48. Singlet Molecular Oxygen

    49. Triplet Molecular Oxygen

    50. Oxygen (8ci,9ci)

    51. Oxygen [vandf]

    52. Oxygen [hsdb]

    53. Oxygen [inci]

    54. Oxygen [jan]

    55. Oxygen (jp17/usp)

    56. Oxygen [who-dd]

    57. Oxygen [who-ip]

    58. Oxygen [mi]

    59. Dioxygen(2.) (triplet)

    60. Oxygen [mart.]

    61. Oxygen, >=99.6%

    62. Oxygen [green Book]

    63. Oxygen, Compressed [un1072] [nonflammable Gas]

    64. Oxygen [ep Monograph]

    65. Oxygen [usp Monograph]

    66. [oo]

    67. Oxygen, >=99.998%

    68. Chembl1234886

    69. Dtxsid2037681

    70. Chebi:26689

    71. Chebi:27140

    72. Oxygenium [who-ip Latin]

    73. Oxygen, Refrigerated Liquid (cryogenic Liquid) [un1073] [nonflammable Gas]

    74. Db09140

    75. Un 1072

    76. Un 1073

    77. Oxygen, Messer(r) Cangas, 99.999%

    78. Ds-014940

    79. (1)o2

    80. E 948

    81. C00007

    82. D00003

    83. O2(2.)

    84. Oxygen, Refrigerated Liquid (cryogenic Liquid)

    85. Oxygen, Compressed [un1072] [nonflammable Gas]

    86. (o2)(..)

    87. (o2)(2.)

    88. Q5203615

    89. Aquanal(tm)-plus Oxygen (o2) 1-12 Mg/l, Refill Pack For 37428

    90. Aquanal(tm)-plus Oxygen (o2) 1-12 Mg/l, Test Set With Color Comparator

    91. 4-chloro-3-(chloro-difluoro-methyl)-4,4-difluoro-1-thiophen-2-yl-but-2-en-2-one

    92. Oxygen, Refrigerated Liquid (cryogenic Liquid) [un1073] [nonflammable Gas]

    2.4 Create Date
    2004-09-16
    3 Chemical and Physical Properties
    Molecular Weight 31.999 g/mol
    Molecular Formula O2
    XLogP3-1.1
    Hydrogen Bond Donor Count0
    Hydrogen Bond Acceptor Count2
    Rotatable Bond Count0
    Exact Mass31.989829239 g/mol
    Monoisotopic Mass31.989829239 g/mol
    Topological Polar Surface Area34.1 Ų
    Heavy Atom Count2
    Formal Charge0
    Complexity0
    Isotope Atom Count0
    Defined Atom Stereocenter Count0
    Undefined Atom Stereocenter Count0
    Defined Bond Stereocenter Count0
    Undefined Bond Stereocenter Count0
    Covalently Bonded Unit Count1
    4 Drug and Medication Information
    4.1 Therapeutic Uses

    Supplemental oxygen is indicated when normal oxygenation is impaired because of pulmonarry injury, which may result from aspiration (chemical pneumonitis) or inhalation of toxic gases. The PO2 shoud be maintained at 70-80 mm Hg or higher if possible.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 490

    Supplemental oxyugen usually is given empirically to patients with altered mental status or suspected hypoxemia.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 490

    Oxygen (100%) is indicated for patients with carbon monoxide poisoning, to increase the conversion of carboxyhemoglobin and carboxymyoblobin to hemoglobin and myoglobin, and to increase oxygen saturation of the plasma and subsequent delivery to tissues.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 490

    Hyperbaric oxygen (HBO) (100%) oxygen delivered to the patient in a pressurized chamber at 2-3 atm of pressure) may be beneficial for patients with severe carbon monoxide (CO) poisoning. It can hasten the reversal of CO binding to hemoglobin and intracellular myoglobin, can provide oxygen independent of hemoglobin, and may have protective actions in reducing postischemic brain damage.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 490

    For more Therapeutic Uses (Complete) data for OXYGEN (16 total), please visit the HSDB record page.

    4.2 Drug Warning

    In paraquat poisoning, oxygen may contribute to lung injury. In fact, slightly hypoxic environments (10-12% oxygen) have been advocated to reduce the risk of pulmonary fibrosis from paraquat.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 491

    Relative contraindications to hyperbaric oxygen therapy include a history of recent middle ear or thoracic surgery, untreated pneumothorax, seizure disorder, and severe sinusitis.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 491

    Prolonged high concentrations of oxygen are associated with pulmonary alveolar tissue damage. In general, the fraction of inspired oxygen (FIO2) shoud not be maintained at greater than 80% for more than 24 hours.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 491

    Oxygen therapy may increase the risk of retrolental fibroplasia in neonates.

    Olson, K.R. (Ed.); Poisoning & Drug Overdose. 5th ed. Lange Medical Books/McGraw-Hill. New York, N.Y. 2007., p. 491

    For more Drug Warnings (Complete) data for OXYGEN (28 total), please visit the HSDB record page.

    4.3 Drug Indication

    Oxygen therapy in clinical settings is used across diverse specialties, including various types of anoxia, hypoxia or dyspnea and any other disease states and conditions that reduce the efficiency of gas exchange and oxygen consumption such as respiratory illnesses, trauma, poisonings and drug overdoses. Oxygen therapy tries to achieve hyperoxia to reduce the extent of hypoxia-induced tissue damage and malfunction.

    For oxygen supplementation and as a carrier gas during inhalation anaesthesia.

    For oxygen supplementation during recovery.

    5 Pharmacology and Biochemistry
    5.1 Pharmacology

    Oxygen therapy improves effective cellular oxygenation, even at a low rate of tissue perfusion. Oxygen molecules adjust hypoxic ventilatory drive by acting on chemoreceptors on carotid bodies that sequentially relay sensory information to the higher processing centers in brainstem. It also attenuates hypoxia-induced mitochondrial depolarization that generates reactive oxygen species and/or apoptosis. Studies investigating on hyperbaric oxygen therapy has shown that oxygen supplementation can induce neural stem cell proliferation in neonatal rats thus promoting neurological regeneration after injuries. CD34+, CD45-dim leukocytes are also potential targets for hyperbaric oxygen therapy benefit as their mobilization was increased in vitro which could facilitate the acceleration of recovery at peripheral sites.

    5.2 ATC Code

    QV03AN01

    V - Various

    V03 - All other therapeutic products

    V03A - All other therapeutic products

    V03AN - Medical gases

    V03AN01 - Oxygen

    5.3 Absorption, Distribution and Excretion

    Route of Elimination

    Exhalation

    During inhalation of normal air the arterial blood leaves lungs about 95% saturated with oxygen, and with a subject standing at rest, the venous blood returns to lungs about 60 to 70% saturated. During 1 min approx 360 cc of oxygen are used up. After forced deep inspiration normal lung vol is about 5 to 5.5 L, 1 L which is O2 ...

    Patty, F. (ed.). Industrial Hygiene and Toxicology: Volume II: Toxicology. 2nd ed. New York: Interscience Publishers, 1963., p. 914

    Arterial blood carries O2 in 2 forms. Most is normally bound to hemoglobin ... A smaller amt is free in soln. The amount of O2 carried ... depends on partial pressure of oxygen. When fully saturated with O2, each g of hemoglobin binds 1.3 vol % of O2. At 37 C, 0.003 vol % O2 is dissolved in blood/torr of partial pressure of O2.

    Gilman, A. G., L. S. Goodman, and A. Gilman. (eds.). Goodman and Gilman's The Pharmacological Basis of Therapeutics. 6th ed. New York: Macmillan Publishing Co., Inc. 1980., p. 325

    Fetal hemoglobin has more affinity for oxygen than maternal hemoglobin under similar conditions of pH & temp. If incompletely oxygen-saturated fetal & maternal blood are allowed to equilibrate across a membrane, partial pressure of O2 will be identical on both sides of membrane, but O2 content of fetal blood ... /will be/ higher ...

    LaDu, B.N., H.G. Mandel, and E.L. Way. Fundamentals of Drug Metabolism and Disposition. Baltimore: Williams and Wilkins, 1971., p. 94

    Oxygen enters the body primarily through the lungs, but may also be taken up by mucous membranes of the GI tract, the middle ear, and the paranasal sinuses. It diffuses from the alveoli into the pulmonary capillaries, dissolves in the blood plasma, enters the red blood cells, and binds to hemoglobin. The red cells transport bound O2 to tissues throughout the body via the circulatory system. In tissues where the partial pressure of O2 is lower than that of the blood, the O2 diffuses out of the red cells, through the capillaries and plasma, and into the cells. As the O2 plasma concentration diminishes, it is replaced by that contained in the red cells. The red blood cells are then circulated back to the lungs in a continuous recycling process ...

    Bingham, E.; Cohrssen, B.; Powell, C.H.; Patty's Toxicology Volumes 1-9 5th ed. John Wiley & Sons. New York, N.Y. (2001)., p. 3:656

    5.4 Metabolism/Metabolites

    ... Most O2 combines with carbon and hydrogen atoms from glucose molecules to form cellular energy, known as adenosine triphosphate or ATP, along with carbon dioxide (CO2) and water. The remaining O2 is combined with various compounds to synthesize cellular structures or elimination products. The CO2 generated in the cells then diffuses back to the red blood cells and returns to the lungs, where it is exhaled. The metabolic water combines with ingested water and the excess is eliminated by excretion through the kidneys or by evaporation from the lungs and skin.

    Bingham, E.; Cohrssen, B.; Powell, C.H.; Patty's Toxicology Volumes 1-9 5th ed. John Wiley & Sons. New York, N.Y. (2001)., p. 3:656

    In the course of O2 metabolism, several toxic substances are generated, including superoxide anion (O2-), hydrogen peroxide (H2O2), hydroxyl radical (OH-), lipid peroxides, and others. Without the availability of several enzymes that destroy these toxic intermediary compounds, cell death quickly occurs. The protective enzymes include superoxide dismutases (SODs), catalase (CAT), and glutathione peroxidase (GP). Glutathione reductase (GR) participates by re-forming glutathione, which is preferentially oxidized, thereby sparing sulfhydryl-bearing proteins and cell wall constituents. Other contributors to the control of oxidant toxicity include vitamin C (ascorbic acid), vitamin E (alpha-tocopherol), vitamin A, and selenium, a cofactor for GP.

    Bingham, E.; Cohrssen, B.; Powell, C.H.; Patty's Toxicology Volumes 1-9 5th ed. John Wiley & Sons. New York, N.Y. (2001)., p. 3:661

    Oxygen (O2) is reduced by both enzymatic and nonenzymatic processes to the superoxide radical (O2-). This radical species of oxygen is postulated to be formed in vivo in animals through activity of some iron-sulfur oxidation-reduction enzymes and certain flavoproteins ...

    Doull, J., C.D. Klaassen, and M. D. Amdur (eds.). Casarett and Doull's Toxicology. 2nd ed. New York: Macmillan Publishing Co., 1980., p. 65

    The partial reduction of molecular oxygen in biological systems produces the cytotoxic intermediates superoxide, hydrogen peroxide, and hydroxyl radical. The superoxide radical is now recognized to play significant roles in a number of pathophysiologic states including oxygen toxicity, radiation damage, phagocyte-mediated inflammation, and postischemic injury.

    McCord JM; The Superoxide Free Radical; Its Biochemistry and Pathophysiology; Surgery 94 (3): 412-4 (1983)

    Oxygen toxicity is mediated through increased production of partially reduced oxygen products such as superoxide anion, perhydroperoxy and hydroxyl radicals, peroxynitrite and possibly singlet molecular oxygen.

    Klaassen, C.D. (ed). Casarett and Doull's Toxicology. The Basic Science of Poisons. 6th ed. New York, NY: McGraw-Hill, 2001., p. 529

    5.5 Biological Half-Life

    Approximately 122.24 seconds

    5.6 Mechanism of Action

    Oxygen therapy increases the arterial pressure of oxygen and is effective in improving gas exchange and oxygen delivery to tissues, provided that there are functional alveolar units. Oxygen plays a critical role as an electron acceptor during oxidative phosphorylation in the electron transport chain through activation of cytochrome c oxidase (terminal enzyme of the electron transport chain). This process achieves successful aerobic respiration in organisms to generate ATP molecules as an energy source in many tissues. Oxygen supplementation acts to restore normal cellular activity at the mitochondrial level and reduce metabolic acidosis. There is also evidence that oxygen may interact with O2-sensitive voltage-gated potassium channels in glomus cells and cause hyperpolarization of mitochondrial membrane.

    The exact mechanism whereby hypoxic pulmonary vasoconstriction is elicited is still unsettled. A possible role for toxic oxygen metabolites was evaluated, employing a set-up of blood-perfused isolated rat lungs. Hypoxic pulmonary vasoconstriction reflected as pulmonary arterial pressor responses, was evoked by alternately challenging the airways with a hypoxic- and a normoxic gas mixture, resulting in gradually increasing responses until a maximum was obtained. In a sequence of responses (mean +/- s.e. mean) increasing from 2.5 +/ - 0.2 kPa to 3.2 +/ - 0.1 kPa, administration to the perfusate of the inhibitor of xanthine oxidase, allopurinol reduced the subsequent response to 2.5 +/- 0.2 kPa (P < 0.001). By contrast, allopurinol did not affect vasoconstriction induced by serotonin or bradykinin. In control experiments responses continued to increase after administration of hypoxanthine (substrate of xanthine oxidase). Neither pretreatment with daily injections of the antioxidant vitamin E for 3 days in advance, nor addition to the perfusate of the scavenger enzymes superoxide dismutase and catalase, or dimethylsulfoxide had any impact on hypoxic pulmonary vasoconstriction; the subsequent responses rose at the same rate and in the same way as before. Thus, the present study has shown that allopurinol inhibition of xanthine oxidase depresses hypoxic pulmonary vasoconstriction. This could be due either to reduced production of toxic oxygen metabolites or to accumulation of purine metabolites. The absence of inhibitory effects of quenchers of toxic oxygen metabolites refutes a role for these metabolites in the elicitation of hypoxic pulmonary vasoconstriction. More likely, allopurinol inhibits hypoxic pulmonary vasoconstriction by interfering with the purine metabolism.

    PMID:2389653 Kjaeve J et al; Acta Anaesthesiol Scand 34 (5): 384-8 (1990)

    Exposure to hyperoxia results in endothelial necrosis followed by type II cell proliferation. This suggests that type II cells are resistant to hyperoxia. Oxygen-induced lung injury may result from an overproduction of oxygen metabolites normally scavenged by antioxidants such as superoxide dismutase, glutathione peroxidase, catalase and reduced glutathione. Therefore, resistance of type II cells to hyperoxia may be linked to high antioxidant activities. To test this hypothesis /the authors/ compared in vitro the effects of a 24 hr exposure period to 95% O2 on cultured type II cells, lung fibroblasts and alveolar macrophages isolated from rats. We show that type II cells, when compared with other cell types, are highly sensitive to hyperoxia as shown by increased lactate dehydrogenase release, decreased deoxyribose nucleic acid and protein content of Petri dishes and decreased thymidine incorporation into DNA. Synthesis of dipalmitoylphosphatidylcholine was also significantly reduced. Antioxidant enzyme activities as well as glutathione content were not higher in type II cells than in other cell types. However, hyperoxia results in a decreased superoxide dismutase activity and glutathione content in type II cells which was not observed in fibroblasts. /It was concluded/ that adaptative changes in superoxide dismutase and glutathione metabolism could be important defense mechanisms in cells exposed to hyperoxia.

    PMID:1756840 Housset B et al; Eur Respir J 4 (9): 1066-75 (1991)

    Oxygen, essential for mammalian life, is paradoxically harmful. If O2 is given at high enough concentrations for long enough times, the body's protective mechanisms are overwhelmed, leading to cellular injury and, with continued exposure, even death. In the course of O2 metabolism, several toxic substances are generated, including superoxide anion (O2-), hydrogen peroxide (H2O2), hydroxyl radical (OH-), lipid peroxides, and others. Without the availability of several enzymes that destroy these toxic intermediary compounds, cell death quickly occurs. The protective enzymes include superoxide dismutases (SODs), catalase (CAT), and glutathione peroxidase (GP). Glutathione reductase (GR) participates by re-forming glutathione, which is preferentially oxidized, thereby sparing sulfhydryl-bearing proteins and cell wall constituents. Other contributors to the control of oxidant toxicity include vitamin C (ascorbic acid), vitamin E (alpha-tocopherol), vitamin A, and selenium, a cofactor for GP. Normally, a balance exists between the production of toxic oxidants and their destruction by antioxidant mechanisms. Some individuals may lack the ability to produce sufficient antioxidants and suffer a slow progressive tissue deterioration as a result.

    Bingham, E.; Cohrssen, B.; Powell, C.H.; Patty's Toxicology Volumes 1-9 5th ed. John Wiley & Sons. New York, N.Y. (2001)., p. 3:662

    Protein accumulation in the BAL fluid results from damage to the pavement-like cells that line the alveolar sacs, known as type I cells, which cover 95% of the alveolar surface. Type I cells are generally incapable of dividing, but when damaged, can be replaced by the type II alveolar cells interspersed among them. Type II cells are less susceptible to toxic injury, can proliferate rapidly, and can be transformed into type I cells. Toxic injuries that affect only type I cells can be repaired by this proliferative process. To the extent that type II cells are also injured, the effects are more severe and may lead to permanent changes. Other types of cells in the lung are also affected, especially the capillary endothelial cells, leading to leakage of blood plasma into the interstitial tissue between the alveoli, and ultimately into the alveoli. Blood cells in the capillaries may also form a clot or may leak into alveolar spaces (hemorrhage). Other cells in the interstitium, such as fibroblasts, are damaged. An inflammatory response, with infiltration of white blood cells, proliferation of fibroblasts, and subsequent fibrosis may follow.

    Bingham, E.; Cohrssen, B.; Powell, C.H.; Patty's Toxicology Volumes 1-9 5th ed. John Wiley & Sons. New York, N.Y. (2001)., p. 3:663

    For more Mechanism of Action (Complete) data for OXYGEN (11 total), please visit the HSDB record page.

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    Rev. Date :

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    AACR Annual meeting
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    Sol S.p.a Group

    Italy

    USDMF, CEP/COS, JDMF, EU-WC, NDC, KDMF, VMF, Others

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    AACR Annual meeting
    Not Confirmed
    USDMF CEP/COS arrow-down
    ✖

    Certificate Number : CEP 2020-295 - Rev 01

    Issue Date : 2026-02-23

    Type : Chemical

    Substance Number : 417

    Status : Valid

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    ASPEN AIR

    Netherlands
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    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205862

    RX/OTC/DISCN :

    RLD :

    TE Code :

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    02

    ABSOLUTE AIR, LLC

    U.S.A
    AACR Annual meeting
    Not Confirmed
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    U.S.A
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 217500

    RX/OTC/DISCN :

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    TE Code :

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    03

    AIR LIQUIDE CANADA INC

    France
    AACR Annual meeting
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    AIR LIQUIDE CANADA INC

    France
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 214857

    RX/OTC/DISCN :

    RLD :

    TE Code :

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    AIR LIQUID INDUSTRIAL US LP

    U.S.A
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    U.S.A
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205712

    RX/OTC/DISCN :

    RLD :

    TE Code :

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    AIR LIQUID INDUSTRIAL US LP

    U.S.A
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205713

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    AIR LIQUIDE LARGE INDUSTRIES US LP

    U.S.A
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    Not Confirmed
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    AIR LIQUIDE LARGE INDUSTRIES US LP

    U.S.A
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205737

    RX/OTC/DISCN :

    RLD :

    TE Code :

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    07

    AIR PRODUCTS AND CHEMICALS INC

    France
    AACR Annual meeting
    Not Confirmed
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    France
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    AACR Annual meeting
    Not Confirmed
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205865

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    TE Code :

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    LINDE NORTH AMERICA INC

    Canada
    AACR Annual meeting
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    Canada
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    AACR Annual meeting
    Not Confirmed
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205767

    RX/OTC/DISCN :

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    TE Code :

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    09

    MATHESON TRI-GAS INC

    U.S.A
    AACR Annual meeting
    Not Confirmed
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    MATHESON TRI-GAS INC

    U.S.A
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    AACR Annual meeting
    Not Confirmed
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205889

    RX/OTC/DISCN :

    RLD :

    TE Code :

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    10

    PRAXAIR INC

    Canada
    AACR Annual meeting
    Not Confirmed
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    Canada
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    AACR Annual meeting
    Not Confirmed
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    RLD :

    TE Code :

    OXYGEN

    Brand Name : OXYGEN, USP

    Dosage Form : GAS;INHALATION

    Dosage Strength : N/A

    Approval Date :

    Application Number : 205849

    RX/OTC/DISCN :

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    Europe

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    01

    AIR LIQUIDE Sanita' Serv. SpA

    France
    AACR Annual meeting
    Not Confirmed
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    AIR LIQUIDE Sanita' Serv. SpA

    France
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 200 Bar 7 Lt Cubic Meters Carried Out 1.54

    Dosage Strength : 200 bar gas compressed med bomb 7 liters

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    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    AIR LIQUIDE Sanita' Serv. SpA

    France
    AACR Annual meeting
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    AIR LIQUIDE Sanita' Serv. SpA

    France
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 200 Bar 11 Lt Cubic Meters Carried Out 2.42

    Dosage Strength : 200 bar gas compressed med bomb 11 liters

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    Regulatory Info :

    Registration Country : Italy

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    AIR LIQUIDE Sanita' Serv. SpA

    France
    AACR Annual meeting
    Not Confirmed
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    AIR LIQUIDE Sanita' Serv. SpA

    France
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Oxygen Gas

    Dosage Form : Oxygen Cryogenic Gas 21,000 Lt Cubic Meters 17966,34 Carried Out

    Dosage Strength : cryogenic med fixed container 21,000 liters

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    04

    LINDE MEDICALE Srl

    Ireland
    AACR Annual meeting
    Not Confirmed
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    LINDE MEDICALE Srl

    Ireland
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    AACR Annual meeting
    Not Confirmed
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Oxygen Gas

    Dosage Form : Oxygen Cryogenic Gas 10,000 Lt Cubic Meters Carried Out 8555,40

    Dosage Strength : cryogenic med fixed container 10,000 liters

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    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    Messer Iberica De Gases, Sau

    Canada
    AACR Annual meeting
    Not Confirmed
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    Messer Iberica De Gases, Sau

    Canada
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    AACR Annual meeting
    Not Confirmed
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    Regulatory Info : Authorized

    Registration Country : Spain

    Oxygen

    Brand Name : Aire Sintetico Medicinal Gas Comprimido Messer

    Dosage Form : Gas For Inhalation In Bottle

    Dosage Strength : 200BAR

    Packaging :

    Approval Date : 09-07-2012

    Application Number : 76170

    Regulatory Info : Authorized

    Registration Country : Spain

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    S.I.A.D. SpA

    Country
    AACR Annual meeting
    Not Confirmed
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    AACR Annual meeting
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 200 Bar 10 Lt Cubic Meters Carried Out 2.2

    Dosage Strength : 200 bar gas compressed med bomb 10 liters

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    MEDIGAS ITALIA Srl

    Country
    AACR Annual meeting
    Not Confirmed
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    Country
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 200 Bar 15 Lt Cubic Meters Carried Out 3.30

    Dosage Strength : 200 bar gas compressed med bomb 15 liters

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    RIVOIRA PHARMA Srl

    Country
    AACR Annual meeting
    Not Confirmed
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    RIVOIRA PHARMA Srl

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    AACR Annual meeting
    Not Confirmed
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 200 Bar 30 Lt Cubic Meters Carried Out 6.60

    Dosage Strength : 200 bar gas compressed med bomb 30 liters

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    S.I.A.D. SpA

    Country
    AACR Annual meeting
    Not Confirmed
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Cryogenic Gas 41 Lt Cubic Meters Carried Out 35,07

    Dosage Strength : cryogenic med mobile container 41 liters

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    MEDICAIR ITALIA Srl

    Country
    AACR Annual meeting
    Not Confirmed
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    Regulatory Info :

    Registration Country : Italy

    Oxygen

    Brand Name : Sigeno

    Dosage Form : Oxygen Gas Compressed 300 Bar 20 Lt Cubic Meters Carried Out 6,30

    Dosage Strength : 300 bar gas compressed med bomb in steel 20 liters liters 6590 carried out with valve vi

    Packaging :

    Approval Date :

    Application Number :

    Regulatory Info :

    Registration Country : Italy

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    ABOUT THIS PAGE

    Looking for 7782-44-7 / Oxygen API manufacturers, exporters & distributors?

    Oxygen manufacturers, exporters & distributors 1

    31

    PharmaCompass offers a list of Oxygen API manufacturers, exporters & distributors, which can be sorted by GMP, USDMF, JDMF, KDMF, CEP (COS), WC, and more, enabling you to easily find the right Oxygen manufacturer or Oxygen supplier for your needs.

    Send us enquiries for free, and we will assist you in establishing a direct connection with your preferred Oxygen manufacturer or Oxygen supplier.

    API | Excipient name

    Oxygen

    Synonyms

    7782-44-7, O, Molecular oxygen, Oxygen molecule, Dioxygen, Pure oxygen

    Cas Number

    7782-44-7

    Unique Ingredient Identifier (UNII)

    S88TT14065

    About Oxygen

    An element with atomic symbol O, atomic number 8, and atomic weight [15.99903; 15.99977]. It is the most abundant element on earth and essential for respiration.

    dioxygene Manufacturers

    A dioxygene manufacturer is defined as any person or entity involved in the manufacture, preparation, processing, compounding or propagation of dioxygene, including repackagers and relabelers. The FDA regulates dioxygene manufacturers to ensure that their products comply with relevant laws and regulations and are safe and effective to use. dioxygene API Manufacturers are required to adhere to Good Manufacturing Practices (GMP) to ensure that their products are consistently manufactured to meet established quality criteria.

    dioxygene Suppliers

    A dioxygene supplier is an individual or a company that provides dioxygene active pharmaceutical ingredient (API) or dioxygene finished formulations upon request. The dioxygene suppliers may include dioxygene API manufacturers, exporters, distributors and traders.

    click here to find a list of dioxygene suppliers with USDMF, JDMF, KDMF, CEP, GMP, and COA related information on PharmaCompass.

    dioxygene USDMF

    A dioxygene DMF (Drug Master File) is a document detailing the whole manufacturing process of dioxygene active pharmaceutical ingredient (API) in detail. Different forms of dioxygene DMFs exist exist since differing nations have different regulations, such as dioxygene USDMF, ASMF (EDMF), JDMF, CDMF, etc.

    A dioxygene DMF submitted to regulatory agencies in the US is known as a USDMF. dioxygene USDMF includes data on dioxygene's chemical properties, information on the facilities and procedures used, and details about packaging and storage. The dioxygene USDMF is kept confidential to protect the manufacturer’s intellectual property.

    click here to find a list of dioxygene suppliers with USDMF on PharmaCompass.

    dioxygene CEP

    A dioxygene CEP of the European Pharmacopoeia monograph is often referred to as a dioxygene Certificate of Suitability (COS). The purpose of a dioxygene CEP is to show that the European Pharmacopoeia monograph adequately controls the purity of dioxygene EP produced by a given manufacturer. Suppliers of raw materials can prove the suitability of dioxygene to their clients by showing that a dioxygene CEP has been issued for it. The manufacturer submits a dioxygene CEP (COS) as part of the market authorization procedure, and it takes on the role of a dioxygene CEP holder for the record. Additionally, the data presented in the dioxygene CEP (COS) is managed confidentially and offers a centralized system acknowledged by numerous nations, exactly like the dioxygene DMF.

    A dioxygene CEP (COS) is recognised by all 36 nations that make up the European Pharmacopoeia Convention. dioxygene CEPs may be accepted in nations that are not members of the Ph. Eur. at the discretion of the authorities there.

    click here to find a list of dioxygene suppliers with CEP (COS) on PharmaCompass.

    dioxygene GMP

    dioxygene Active pharmaceutical ingredient (API) is produced in GMP-certified manufacturing facility.

    GMP stands for Good Manufacturing Practices, which is a system used in the pharmaceutical industry to make sure that goods are regularly produced and monitored in accordance with quality standards. The FDA’s current Good Manufacturing Practices requirements are referred to as cGMP or current GMP which indicates that the company follows the most recent GMP specifications. The World Health Organization (WHO) has its own set of GMP guidelines, called the WHO GMP. Different countries can also set their own guidelines for GMP like China (Chinese GMP) or the EU (EU GMP).

    PharmaCompass offers a list of dioxygene GMP manufacturers, exporters & distributors, which can be sorted by USDMF, JDMF, KDMF, CEP (COS), WC, and more, enabling you to easily find the right dioxygene GMP manufacturer or dioxygene GMP API supplier for your needs.

    dioxygene CoA

    A dioxygene CoA (Certificate of Analysis) is a formal document that attests to dioxygene's compliance with dioxygene specifications and serves as a tool for batch-level quality control.

    dioxygene CoA mostly includes findings from lab analyses of a specific batch. For each dioxygene CoA document that a company creates, the USFDA specifies specific requirements, such as supplier information, material identification, transportation data, evidence of conformity and signature data.

    dioxygene may be tested according to a variety of international standards, such as European Pharmacopoeia (dioxygene EP), dioxygene JP (Japanese Pharmacopeia) and the US Pharmacopoeia (dioxygene USP).

    Inform the supplier about your product requirements, specifying if you need a product with particular monograph like EP (Ph. Eur.), USP, JP, BP, or any other quality. In addition, clarify whether you need hydrochloride (HCl), anhydricum, base, micronisatum or a specific level of purity. To find reputable suppliers, utilize the filters and select those certified by GMP, FDA, or any other certification as per your requirement.
    For your convenience, we have listed synonyms and CAS numbers to help you find the best supplier. The use of synonyms and CAS numbers can be helpful in identifying potential suppliers, but it is crucial to note that they might not always indicate the exact same product. It is important to confirm the product details with the supplier before making a purchase to ensure that it meets your requirements.
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