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Chemistry

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Also known as: 61869-08-7, Paxil, Frosinor, Paxil cr, Casbol, Motivan
Molecular Formula
C19H20FNO3
Molecular Weight
329.4  g/mol
InChI Key
AHOUBRCZNHFOSL-YOEHRIQHSA-N
FDA UNII
32Q7TW8BX7

Paroxetine Hydrochloride
A serotonin uptake inhibitor that is effective in the treatment of depression.
Paroxetine is a Serotonin Reuptake Inhibitor. The mechanism of action of paroxetine is as a Serotonin Uptake Inhibitor.
1 2D Structure

Paroxetine Hydrochloride

2 Identification
2.1 Computed Descriptors
2.1.1 IUPAC Name
(3S,4R)-3-(1,3-benzodioxol-5-yloxymethyl)-4-(4-fluorophenyl)piperidine
2.1.2 InChI
InChI=1S/C19H20FNO3/c20-15-3-1-13(2-4-15)17-7-8-21-10-14(17)11-22-16-5-6-18-19(9-16)24-12-23-18/h1-6,9,14,17,21H,7-8,10-12H2/t14-,17-/m0/s1
2.1.3 InChI Key
AHOUBRCZNHFOSL-YOEHRIQHSA-N
2.1.4 Canonical SMILES
C1CNCC(C1C2=CC=C(C=C2)F)COC3=CC4=C(C=C3)OCO4
2.1.5 Isomeric SMILES
C1CNC[C@H]([C@@H]1C2=CC=C(C=C2)F)COC3=CC4=C(C=C3)OCO4
2.2 Other Identifiers
2.2.1 UNII
32Q7TW8BX7
2.3 Synonyms
2.3.1 MeSH Synonyms

1. Aropax

2. Brl 29060

3. Brl-29060

4. Brl29060

5. Fg 7051

6. Fg-7051

7. Fg7051

8. Paroxetine Acetate

9. Paroxetine Hydrochloride

10. Paroxetine Hydrochloride Anhydrous

11. Paroxetine Hydrochloride Hemihydrate

12. Paroxetine Hydrochloride, Hemihydrate

13. Paroxetine Maleate

14. Paroxetine, Cis-(+)-isomer

15. Paroxetine, Cis-(-)-isomer

16. Paroxetine, Trans-(+)-isomer

17. Paxil

18. Seroxat

2.3.2 Depositor-Supplied Synonyms

1. 61869-08-7

2. Paxil

3. Frosinor

4. Paxil Cr

5. Casbol

6. Motivan

7. Paxpar

8. Seroxat

9. Paroxetina

10. Paroxetinum

11. Aropax

12. Paroxetinum [inn-latin]

13. Paroxetina [inn-spanish]

14. Brl 29060

15. Paxetil

16. Brl-29060

17. (-)-paroxetine

18. Fg 7051

19. Fg-7051

20. (3s,4r)-3-((benzo[d][1,3]dioxol-5-yloxy)methyl)-4-(4-fluorophenyl)piperidine

21. Arketis

22. Besitram

23. Daparox

24. Parogen

25. Xetanor

26. Arotin

27. (3s,4r)-3-[(1,3-benzodioxol-5-yloxy)methyl]-4-(4-fluorophenyl)piperidine

28. (-)-(3s,4r)-4-(p-fluorophenyl)-3-((3,4-(methylenedioxy)phenoxy)methyl)piperidine

29. [3h]paroxetine

30. (+/-)-paroxetine

31. (-)3s,4r-paroxetine

32. (3s,4r)-3-(1,3-benzodioxol-5-yloxymethyl)-4-(4-fluorophenyl)piperidine

33. Paroxetine, (+/-)-

34. Chembl490

35. Paroxetine, Trans-(+/-)-

36. Piperidine, 3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)-, (3s,4r)-

37. 32q7tw8bx7

38. Chebi:7936

39. 41vrh5220h

40. (-)-(3s,4r)-4-(p-fluorophenyl)-3-((3,4-methylenedioxy)phenoxy)methyl)piperidine

41. Piperidine, 3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)-, (3s-trans)-

42. Dsstox_cid_3425

43. (3s,4r)-3-[(2h-1,3-benzodioxol-5-yloxy)methyl]-4-(4-fluorophenyl)piperidine

44. (3s-trans)-3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)piperidine

45. 110429-35-1

46. Paroxetine (tn)

47. Dsstox_rid_77022

48. Dsstox_gsid_23425

49. Paroxetine [usan:inn:ban]

50. Chembl1708

51. Piperidine, 3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)-, (3r,4s)-rel-

52. Piperidine, 3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)-, Trans-

53. Piperidine, 3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)-, Trans-(+/-)-

54. Rel-(3r,4s)-3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)piperidine

55. Paroxetine Hcl Hemihydrate

56. 130855-15-1

57. Cas-61869-08-7

58. Paroxetine (usp/inn)

59. Unii-41vrh5220h

60. 3h-paroxetine

61. Paroxetine.hcl

62. (3s,4r)-3-((1,3-benzodioxol-5-yloxy)methyl)-4-(4-fluorophenyl)piperidine

63. Piperidine, 3-[(1,3-benzodioxol-5-yloxy)methyl]-4-(4-fluorophenyl)-, (3s,4r)-

64. Piperidine, 3-[(1,3-benzodioxol-5-yloxy)methyl]-4-(4-fluorophenyl)-, (3s-trans)-

65. Ncgc00182968-01

66. 8pr

67. Nnc-20-7051

68. Spectrum_001752

69. Paroxetine [mi]

70. Specplus_000788

71. Paroxetine [inn]

72. Prestwick3_000851

73. Spectrum5_001665

74. Paroxetine [usan]

75. Paroxetine [vandf]

76. Paroxetine [mart.]

77. Paroxetine [who-dd]

78. Unii-32q7tw8bx7

79. Schembl27799

80. Bspbio_000861

81. Kbioss_002232

82. Bidd:gt0673

83. Divk1c_006884

84. Bpbio1_000949

85. Gtpl4790

86. Dtxsid3023425

87. Bdbm22416

88. Hsdb 7175

89. Kbio1_001828

90. Kbio2_002232

91. Kbio2_004800

92. Kbio2_007368

93. Hms2090h05

94. Zinc527386

95. Tox21_113123

96. Bdbm50331515

97. (-)-trans-4-(4-fluorophenyl)-3-(3,4-methylenedioxyphenoxymethyl)piperidine

98. Akos015888636

99. Tox21_113123_1

100. Ac-8185

101. Db00715

102. Sdccgsbi-0051908.p003

103. (-)-trans-4-(p-fluorophenyl)-3-[[3,4-(methylenedioxy)phenoxy]methyl]-piperidine

104. (3s-trans)-3-[(1,3-benzodioxol-5-yl-oxy)methyl]-4-(4-fluorophenyl)piperidine

105. Ncgc00025355-02

106. Ncgc00025355-03

107. Ncgc00025355-04

108. Ncgc00025355-05

109. Ncgc00025355-06

110. Ncgc00025355-07

111. Ncgc00025355-08

112. Ncgc00025355-09

113. Ncgc00025355-12

114. Ncgc00025355-22

115. Sbi-0051908.p002

116. Hy-122272

117. Ab00514724

118. Cs-0083299

119. C07415

120. D02362

121. Ab00053704-21

122. Ab00053704_22

123. Ab00053704_23

124. Paroxetine Hydrochloride Anhydrous Ep Impurity E

125. 869p087

126. Paroxetine Hydrochloride (anhydrous Or Hemihydrate)

127. Q408471

128. Brd-k37991163-003-02-7

129. Brd-k37991163-050-05-1

130. (-)-trans-4-(p-fluorophenyl)-3-((3,4-(methylenedioxy)phenoxy)methyl)piperidine

131. Piperidine, 3-[(1,3-benzodioxol-5-yloxy)methyl]-4-(4-fluorophenyl)-,(3s,4r)-

132. Cis-paroxetine; Paroxetine Usp Related Compound D;[(3s,4r)-4-(p-fluorophenyl)-3-piperidyl](1,3-dioxa-5-indanyloxy)methane

2.4 Create Date
2005-06-08
3 Chemical and Physical Properties
Molecular Weight 329.4 g/mol
Molecular Formula C19H20FNO3
XLogP33.5
Hydrogen Bond Donor Count1
Hydrogen Bond Acceptor Count5
Rotatable Bond Count4
Exact Mass329.14272166 g/mol
Monoisotopic Mass329.14272166 g/mol
Topological Polar Surface Area39.7 Ų
Heavy Atom Count24
Formal Charge0
Complexity402
Isotope Atom Count0
Defined Atom Stereocenter Count2
Undefined Atom Stereocenter Count0
Defined Bond Stereocenter Count0
Undefined Bond Stereocenter Count0
Covalently Bonded Unit Count1
4 Drug and Medication Information
4.1 Drug Information
1 of 6  
Drug NamePaxil
PubMed HealthParoxetine (By mouth)
Drug ClassesAntidepressant, Central Nervous System Agent
Drug LabelDESCRIPTIONPAXIL CR (paroxetine hydrochloride) is an orally administered psychotropic drug with a chemical structure unrelated to other selective serotonin reuptake inhibitors or to tricyclic, tetracyclic, or other available antidepressant or antipan...
Active IngredientParoxetine hydrochloride
Dosage FormTablet; Suspension
RouteOral
Strengtheq 20mg base; eq 40mg base; eq 10mg base/5ml; eq 30mg base; eq 10mg base
Market StatusPrescription
CompanyApotex Technologies

2 of 6  
Drug NamePaxil cr
Drug LabelPAXIL CR (paroxetine hydrochloride) is an orally administered psychotropic drug with a chemical structure unrelated to other selective serotonin reuptake inhibitors or to tricyclic, tetracyclic, or other available antidepressant or antipanic agents....
Active IngredientParoxetine hydrochloride
Dosage FormTablet, extended release
RouteOral
Strengtheq 12.5mg base; eq 25mg base; eq 37.5mg base
Market StatusPrescription
CompanyApotex Technologies

3 of 6  
Drug NamePexeva
Active IngredientParoxetine mesylate
Dosage FormTablet
RouteOral
Strengtheq 20mg base; eq 40mg base; eq 30mg base; eq 10mg base
Market StatusPrescription
CompanyNoven Therap

4 of 6  
Drug NamePaxil
PubMed HealthParoxetine (By mouth)
Drug ClassesAntidepressant, Central Nervous System Agent
Drug LabelDESCRIPTIONPAXIL CR (paroxetine hydrochloride) is an orally administered psychotropic drug with a chemical structure unrelated to other selective serotonin reuptake inhibitors or to tricyclic, tetracyclic, or other available antidepressant or antipan...
Active IngredientParoxetine hydrochloride
Dosage FormTablet; Suspension
RouteOral
Strengtheq 20mg base; eq 40mg base; eq 10mg base/5ml; eq 30mg base; eq 10mg base
Market StatusPrescription
CompanyApotex Technologies

5 of 6  
Drug NamePaxil cr
Drug LabelPAXIL CR (paroxetine hydrochloride) is an orally administered psychotropic drug with a chemical structure unrelated to other selective serotonin reuptake inhibitors or to tricyclic, tetracyclic, or other available antidepressant or antipanic agents....
Active IngredientParoxetine hydrochloride
Dosage FormTablet, extended release
RouteOral
Strengtheq 12.5mg base; eq 25mg base; eq 37.5mg base
Market StatusPrescription
CompanyApotex Technologies

6 of 6  
Drug NamePexeva
Active IngredientParoxetine mesylate
Dosage FormTablet
RouteOral
Strengtheq 20mg base; eq 40mg base; eq 30mg base; eq 10mg base
Market StatusPrescription
CompanyNoven Therap

4.2 Therapeutic Uses

Antidepressive Agents, Second-Generation; Serotonin Uptake Inhibitors

National Library of Medicine's Medical Subject Headings. Paroxetine. Online file (MeSH, 2015). Available from, as of May 1, 2015: https://www.nlm.nih.gov/mesh/2014/mesh_browser/MBrowser.html


/CLINICAL TRIALS/ ClinicalTrials.gov is a registry and results database of publicly and privately supported clinical studies of human participants conducted around the world. The Web site is maintained by the National Library of Medicine (NLM) and the National Institutes of Health (NIH). Each ClinicalTrials.gov record presents summary information about a study protocol and includes the following: Disease or condition; Intervention (for example, the medical product, behavior, or procedure being studied); Title, description, and design of the study; Requirements for participation (eligibility criteria); Locations where the study is being conducted; Contact information for the study locations; and Links to relevant information on other health Web sites, such as NLM's MedlinePlus for patient health information and PubMed for citations and abstracts for scholarly articles in the field of medicine. Paroxetine is included in the database.

NIH/NLM; ClinicalTrials.Gov. Available from, as of July 18, 2015: https://clinicaltrials.gov/search/intervention=paroxetine


Paxil is indicated for the treatment of major depressive disorder. /Included in US product labeling/

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


Paxil is indicated for the treatment of obsessions and compulsions in patients with obsessive compulsive disorder (OCD) as defined in the DSM-IV. The obsessions or compulsions cause marked distress, are time-consuming, or significantly interfere with social or occupational functioning. /Included in US product labeling/

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


For more Therapeutic Uses (Complete) data for PAROXETINE (13 total), please visit the HSDB record page.


4.3 Drug Warning

/BOXED WARNING/ SUICIDALITY AND ANTIDEPRESSANT DRUGS. Antidepressants increased the risk compared to placebo of suicidal thinking and behavior (suicidality) in children, adolescents, and young adults in short-term studies of major depressive disorder (MDD) and other psychiatric disorders. Anyone considering the use of Paxil or any other antidepressant in a child, adolescent, or young adult must balance this risk with the clinical need. Short-term studies did not show an increase in the risk of suicidality with antidepressants compared to placebo in adults beyond age 24; there was a reduction in risk with antidepressants compared to placebo in adults aged 65 and older. Depression and certain other psychiatric disorders are themselves associated with increases in the risk of suicide. Patients of all ages who are started on antidepressant therapy should be monitored appropriately and observed closely for clinical worsening, suicidality, or unusual changes in behavior. Families and caregivers should be advised of the need for close observation and communication with the prescriber. Paxil is not approved for use in pediatric patients.

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


/BOXED WARNING/ WARNING: SUICIDAL THOUGHTS AND BEHAVIORS. Antidepressants, including selective serotonin reuptake inhibitors (SSRIs), have been shown to increase the risk of suicidal thoughts and behavior in pediatric and young adult patients when used to treat major depressive disorder and other psychiatric disorders. Because Brisdelle is an SSRI, monitor patients closely for worsening and for emergence of suicidal thoughts and behaviors. Advise families and caregivers of the need for close observation and communication with the prescriber.

NIH; DailyMed. Current Medication Information for Brisdelle (Paroxetine Mesylate) Capsule (Updated: December 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=bf208751-e6d8-11e1-aff1-0800200c9a66


Somnolence, which appears to be dose related, is among the most common adverse effects of paroxetine, occurring in approximately 23% of depressed patients receiving the drug in short-term controlled clinical trials. Somnolence required discontinuance of therapy in about 2% of patients. Headache occurred in about 18 or 15% of patients receiving paroxetine in short- or long-term controlled clinical trials, respectively. In addition, migraine or vascular headache has been reported in up to 1% or less than 0.1% of paroxetine-treated patients, respectively. Asthenia, which also appears to be dose related,1 occurred in 15% of depressed patients receiving the drug in short-term controlled clinical trials and required discontinuance of therapy in about 2% of patients.

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2348


Dizziness, which appears to be dose related, occurred in about 13% of patients receiving paroxetine in short-term controlled clinical trials. Insomnia occurred in about 13 or 8% of patients receiving the drug in short- or long-term controlled clinical trials, respectively. However, because insomnia is a symptom also associated with depression, relief of insomnia and improvement in sleep patterns may occur when clinical improvement in depression becomes apparent during antidepressant therapy. In clinical trials, less than 2% of patients discontinued paroxetine because of insomnia.

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2348


For more Drug Warnings (Complete) data for PAROXETINE (41 total), please visit the HSDB record page.


4.4 Drug Indication

Paroxetine is indicated for the management of depression, obsessive-compulsive disorder, panic disorder, social anxiety disorder, generalized anxiety disorder, posttraumatic stress disorder. One form of paroxetine, commercially known as Brisdelle, is used to manage mild to moderate vasomotor symptoms of menopause. Off-label, paroxetine may be used for the treatment of premature ejaculation or irritable bowel syndrome (IBS).


5 Pharmacology and Biochemistry
5.1 Pharmacology

Paroxetine treats the symptoms of depression, various anxiety disorders, posttraumatic stress disorder, obsessive-compulsive disorder, and the vasomotor symptoms of menopause via the inhibition of serotonin reuptake. The onset of action of paroxetine is reported to be approximately 6 weeks. Due its serotonergic activity, paroxetine, like other SSRI drugs, may potentiate serotonin syndrome. This risk is especially high when monoamine oxidase (MAO) inhibitors are given within 2 weeks of paroxetine administration. Upon cessation of MAO inhibitors, a 2-week interval before paroxetine administration is recommended. Do not coadminister these agents.


5.2 MeSH Pharmacological Classification

Cytochrome P-450 CYP2D6 Inhibitors

Drugs and compounds which inhibit or antagonize the biosynthesis or actions of CYTOCHROME P-450 CYP2D6. (See all compounds classified as Cytochrome P-450 CYP2D6 Inhibitors.)


Selective Serotonin Reuptake Inhibitors

Compounds that specifically inhibit the reuptake of serotonin in the brain. (See all compounds classified as Selective Serotonin Reuptake Inhibitors.)


Antidepressive Agents, Second-Generation

A structurally and mechanistically diverse group of drugs that are not tricyclics or monoamine oxidase inhibitors. The most clinically important appear to act selectively on serotonergic systems, especially by inhibiting serotonin reuptake. (See all compounds classified as Antidepressive Agents, Second-Generation.)


5.3 FDA Pharmacological Classification
5.3.1 Active Moiety
PAROXETINE
5.3.2 FDA UNII
41VRH5220H
5.3.3 Pharmacological Classes
Serotonin Uptake Inhibitors [MoA]; Serotonin Reuptake Inhibitor [EPC]
5.4 ATC Code

N - Nervous system

N06 - Psychoanaleptics

N06A - Antidepressants

N06AB - Selective serotonin reuptake inhibitors

N06AB05 - Paroxetine


5.5 Absorption, Distribution and Excretion

Absorption

Paroxetine is readily absorbed from the gastrointestinal tract. Due to the first-pass metabolism, the bioavailability ranges from 30-60%. Cmax is attained 2 to 8 hours after an oral dose. Mean Tmax is 4.3 hours in healthy patients. The steady-state concentration of paroxetine is achieved within 7 to 14 days of oral therapy. In a pharmacokinetic study, AUC in healthy patients was 574 ngh/mL and 1053 ngh/mL in those with moderate renal impairment.


Route of Elimination

About 2/3 of a single paroxetine dose is found to be excreted in the urine and the remainder is found to be excreted in feces. Almost all of the dose is eliminated as metabolites; 3% is found to be excreted as unchanged paroxetine. About 64% of a 30 mg oral dose was found excreted in the urine, with 2% as the parent drug and 62% appearing as metabolites. Approximately 36% of the dose was found to be eliminated in the feces primarily as metabolites and less than 1% as the parent compound.


Volume of Distribution

Paroxetine has a large volume of distribution and is found throughout the body, including in the central nervous system. Only 1% of the drug is found in the plasma. Paroxetine is found in the breast milk at concentrations similar to the concentrations found in plasma.


Clearance

The apparent oral clearance of paroxetine is 167 L/h. The clearance of paroxetine in patients with renal failure is significantly lower and dose adjustment may be required, despite the fact that it is mainly cleared by the liver. Dose adjustments may be required in hepatic impairment.


Paroxetine hydrochloride appears to be slowly but well absorbed from the GI tract following oral administration. Although the oral bioavailability of paroxetine hydrochloride in humans has not been fully elucidated to date, the manufacturer states that paroxetine is completely absorbed after oral dosing of a solution of the hydrochloride salt. However, the relative proportion of an oral dose that reaches systemic circulation unchanged appears to be relatively small because paroxetine undergoes extensive first-pass metabolism. The oral tablets and suspension of paroxetine hydrochloride reportedly are bioequivalent.

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2359


In steady-state dose proportionality studies involving elderly and nonelderly patients, at doses of 20 mg to 40 mg daily for the elderly and 20 mg to 50 mg daily for the nonelderly, some nonlinearity was observed in both populations, again reflecting a saturable metabolic pathway. In comparison to Cmin values after 20 mg daily, values after 40 mg daily were only about 2 to 3 times greater than doubled.

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


Approximately 95% and 93% of paroxetine is bound to plasma protein at 100 ng/mL and 400 ng/mL, respectively. Under clinical conditions, paroxetine concentrations would normally be less than 400 ng/mL. Paroxetine does not alter the in vitro protein binding of phenytoin or warfarin.

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


Paroxetine distributes throughout the body, including the CNS, with only 1% remaining in the plasma.

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


For more Absorption, Distribution and Excretion (Complete) data for PAROXETINE (13 total), please visit the HSDB record page.


5.6 Metabolism/Metabolites

Paroxetine metabolism occurs in the liver and is largely mediated by cytochrome CYP2D6 with contributions from CYP3A4 and possibly other cytochrome enzymes. Genetic polymorphisms of the CYP2D6 enzyme may alter the pharmacokinetics of this drug. Poor metabolizers may demonstrate increased adverse effects while rapid metabolizers may experience decreased therapeutic effects. The majority of a paroxetine dose is oxidized to a catechol metabolite that is subsequently converted to both glucuronide and sulfate metabolites via methylation and conjugation. In rat synaptosomes, the glucuronide and sulfate conjugates have been shown to thousands of times less potent than paroxetine itself. The metabolites of paroxetine are considered inactive.


The exact metabolic fate of paroxetine has not been fully elucidated; however, paroxetine is extensively metabolized, probably in the liver. The principal metabolites are polar and conjugated products of oxidation and methylation, which are readily cleared by the body. Conjugates with glucuronic acid and sulfate predominate, and the principal metabolites have been isolated and identified. The metabolites of paroxetine have been shown to possess no more than 2% of the potency of the parent compound as inhibitors of serotonin reuptake; therefore, they are essentially inactive.

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2359


Paroxetine is extensively metabolized after oral administration. The principal metabolites are polar and conjugated products of oxidation and methylation, which are readily cleared. Conjugates with glucuronic acid and sulfate predominate, and major metabolites have been isolated and identified. Data indicate that the metabolites have no more than 1/50 the potency of the parent compound at inhibiting serotonin uptake. The metabolism of paroxetine is accomplished in part by CYP2D6. Saturation of this enzyme at clinical doses appears to account for the nonlinearity of paroxetine kinetics with increasing dose and increasing duration of treatment. The role of this enzyme in paroxetine metabolism also suggests potential drug-drug interactions

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


Paroxetine has known human metabolites that include 4-[[(3S,4R)-4-(4-Fluorophenyl)piperidin-3-yl]methoxy]benzene-1,2-diol.

S73 | METXBIODB | Metabolite Reaction Database from BioTransformer | DOI:10.5281/zenodo.4056560


5.7 Biological Half-Life

The mean elimination half-life of paroxetine is about 21 hours. In healthy young subjects, mean elimination half-life was found to be 17.3 hours.


Paroxetine hydrochloride is completely absorbed after oral dosing of a solution of the hydrochloride salt. In a study in which normal male subjects (n = 15) received 30 mg tablets daily for 30 days, steady-state paroxetine concentrations were achieved by approximately 10 days for most subjects, although it may take substantially longer in an occasional patient. At steady state, mean ... half life was ... 21.0 hours (CV 32%) ... .

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


The mean elimination half-life is approximately 21 hours (CV 32%) after oral dosing of 30 mg tablets daily for 30 days of Paxil.

NIH; DailyMed. Current Medication Information for Paxil (Paroxetine Hydrochloride Hemihydrate) Tablet, Film Coated; Paxil (Paroxetine Hydrochloride Hemihydrate) Suspension (Updated: July 2014). Available from, as of May 1, 2015: https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=584ace29-6e40-432f-950f-ab7e98653d32


The elimination half-life of paroxetine when administered as paroxetine hydrochloride averages approximately 21-24 hours, although there is wide interpatient variation with half-lives (ranging from 7-65 hours in one study). In healthy males receiving one 30-mg tablet of paroxetine (administered as paroxetine mesylate) once daily for 24 days, the mean paroxetine half-life was 33.2 hours. In geriatric individuals, elimination half-life of paroxetine (administered as paroxetine hydrochloride) may be increased (e.g., to about 36 hours).

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2359


5.8 Mechanism of Action

Paroxetine enhances serotonergic activity via the inhibition presynaptic reuptake of serotonin by the serotonin (SERT) receptor. This inhibition raises the level of serotonin in the synaptic cleft, relieving various symptoms. This drug has been demonstrated to be a stronger inhibitor of serotonin reuptake than other members of the same drug class, including [Citalopram], [Fluoxetine], and [Fluvoxamine]. The mechanism of action of paroxetine in relieving the vasomotor symptoms of menopause is unknown, according to the Brisdelle prescribing information, but may occur due to its effects on thermoregulation. Paroxetine shows a clinically insignificant affinity for adrenergic alpha-1 and alpha-2 receptors and -adrenergic receptors, dopamine D1 and D2 receptors, histamine H1 receptors and serotonin 5-HT1A, 5-HT2A and 5-HT2C receptors. This drug shows some affinity for muscarinic cholinergic receptors and 5-H2B receptors. The delayed onset of paroxetine therapeutic effects may be explained by the initial paroxetine actions on the 5-HT neurons. In rats, paroxetine activates 5-HT1A receptors when it is first administered, inhibiting the stimulation of the 5-HT neurons and subsequent release of serotonin at the synaptic cleft.


Functional and structural approaches were used to examine the inhibitory mechanisms and binding site location for fluoxetine and paroxetine, two serotonin selective reuptake inhibitors, on nicotinic acetylcholine receptors (AChRs) in different conformational states. The results establish that: (a) fluoxetine and paroxetine inhibit h alpha1beta1 gammadelta AChR-induced Ca(2+) influx with higher potencies than dizocilpine. The potency of fluoxetine is increased approximately 10-fold after longer pre-incubation periods, which is in agreement with the enhancement of (3)H-cytisine binding to resting but activatable Torpedo AChRs elicited by these antidepressants, (b) fluoxetine and paroxetine inhibit the binding of the phencyclidine analog piperidyl-3,4-(3)H(N)]-(N-(1-(2 thienyl)cyclohexyl)-3,4-piperidine to the desensitized Torpedo AChR with higher affinities compared to the resting AChR, and (c) fluoxetine inhibits (3)H-dizocilpine binding to the desensitized AChR, suggesting a mutually exclusive interaction. This is supported by our molecular docking results where neutral dizocilpine and fluoxetine and the conformer of protonated fluoxetine with the highest LUDI score interact with the domain between the valine (position 13') and leucine (position 9') rings. Molecular mechanics calculations also evidence electrostatic interactions of protonated fluoxetine at positions 20', 21', and 24'. Protonated dizocilpine bridges these two binding domains by interacting with the valine and outer (position 20') rings. The high proportion of protonated fluoxetine and dizocilpine calculated at physiological pH suggests that the protonated drugs can be attracted to the channel mouth before binding deeper within the AChR ion channel between the leucine and valine rings, a domain shared with phencyclidine, finally blocking ion flux and inducing AChR desensitization.

PMID:20079457 Arias HR et al; Int J Biochem Cell Biol 42 (5): 712-24 (2010)


Paroxetine was shown to be a potent (Ki = 1.1 nM) and specific inhibitor of [3H]-5-hydroxytryptamine (5-HT) uptake into rat cortical and hypothalamic synaptosomes in vitro. Lineweaver-Burk kinetic analysis determined that this inhibition was competitive in nature, implying a direct interaction with the 5-HT uptake transporter complex. Oral administration of paroxetine produced a dose-related inhibition of [3H]-5-HT uptake (ED50 = 1.9 mg/kg) into rat hypothalamic synaptosomes ex vivo with little effect on [3H]-l-noradrenaline (NA) uptake (ED50 greater than 30 mg/kg). This selectivity for 5-HT uptake was maintained after oral dosing for 14 days. Paroxetine (ED50 1-3 mg/kg PO) prevented the 5-HT depleting effect of p-chloroamphetamine (PCA) in rat brain, demonstrating 5-HT uptake blockade in vivo. Radioligand binding techniques in rat brain in vitro showed that paroxetine has little affinity for alpha 1, alpha 2 or beta adrenoceptors, dopamine (D2), 5-HT1, 5-HT2 or histamine (H1) receptors at concentrations below 1000 nM. Paroxetine demonstrated weak affinity for muscarinic receptors (Ki = 89 nM) but was at least 15 fold weaker than amitriptyline (Ki = 5.1 nM). Paroxetine, therefore, provides a useful pharmacological tool for investigating 5-HT systems and furthermore should be an antidepressant with reduced tricyclic-like side-effects.

PMID:2962217 Thomas DR et al; Psychopharmacology (Berl) 93 (2): 193-200 (1987)


The precise mechanism of antidepressant action of paroxetine is unclear, but the drug has been shown to selectively inhibit the reuptake of serotonin at the presynaptic neuronal membrane. Paroxetine-induced inhibition of serotonin reuptake causes increased synaptic concentrations of serotonin in the CNS, resulting in numerous functional changes associated with enhanced serotonergic neurotransmission. Like other SSRIs (e.g., citalopram, fluoxetine, fluvoxamine, sertraline), paroxetine appears to have only very weak effects on the reuptake of norepinephrine or dopamine and does not exhibit clinically important anticholinergic, antihistaminic, or adrenergic (a1, a2, beta) blocking activity at usual therapeutic dosages. Although the mechanism of antidepressant action of antidepressant agents may involve inhibition of the reuptake of various neurotransmitters (i.e., serotonin, norepinephrine) at the presynaptic neuronal membrane, it has been suggested that postsynaptic receptor modification is mainly responsible for the antidepressant action observed during long-term administration of antidepressant agents. During long-term therapy with most antidepressants (e.g., tricyclic antidepressants, monoamine oxidase (MAO) inhibitors), these adaptive changes mainly consist of subsensitivity of the noradrenergic adenylate cyclase system in association with a decrease in the number of beta-adrenergic receptors; such effects on noradrenergic receptor function are commonly referred to as down regulation. However, in an animal study, long-term administration of paroxetine was not shown to downregulate noradrenergic receptors in the CNS as has been observed with many other clinically effective antidepressants. In addition, some antidepressants (e.g., amitriptyline) reportedly decrease the number of serotonergic (5-HT) binding sites following chronic administration.

American Society of Health-System Pharmacists 2015; Drug Information 2015. Bethesda, MD. 2015, p. 2357


Reduced glucose metabolism has been implicated as a pathophysiology of depressive disorder. Normalization of such impaired neurometabolism has been related to the therapeutic actions of antidepressant medication. However, the molecular mechanism underlying the neurometabolic actions of antidepressants has not been fully understood. Given that AMP-activated protein kinase (AMPK) is a master switch for energy homeostasis, we aimed to determine whether selective serotonin reuptake inhibitor paroxetine enhances energy metabolism by activating AMPK in neuroblastoma cells. We found that paroxetine dose dependently increased mitochondrial biogenesis, which involves the AMPK-peroxisome proliferator-activated receptor-gamma coactivator-1a pathway. In addition, paroxetine-induced AMPK activation increases glucose uptake and ATP production. These neurometabolic effects of paroxetine were suppressed by cotreatment with compound C (CC), an AMPK inhibitor. These findings suggest a possibility that modulation of the AMPK pathway might be a previously unrecognized mechanism underlying the neurometabolic action of antidepressants. Further study is warranted to examine the region-specific and time-specific effects of AMPK modulation by antidepressants on mood-related behaviors.

PMID:25839176 Jeong J et al; Neuroreport 26 (7): 424-8 (2015)


For more Mechanism of Action (Complete) data for PAROXETINE (9 total), please visit the HSDB record page.


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MFG.DT. 05\/12\/2023 RETEST DT. 04\/12\/2029","address":"PLOT NO.5222, PHASE- IV,","city":"AHMEDABAD -","supplier":"ZHEJIANG HUAHAI PHARMACEUTICAL","supplierCountry":"CHINA","foreign_port":"SHANGHAI","customer":"UNISON PHARMACEUTICALS PRIVATE LIMITED","customerCountry":"INDIA","quantity":"40.00","actualQuantity":"40","unit":"KGS","unitRateFc":"660","totalValueFC":"26672.5","currency":"USD","unitRateINR":"55374","date":"26-Feb-2024","totalValueINR":"2214960","totalValueInUsd":"26672.5","indian_port":"Ahmedabad Air","hs_no":"29420090","bill_no":"2308068","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"SHANGHAI","supplierAddress":"XUNQIAO,LINHAI ,ZHEJIANG 317024 CHISDNF CHINA","customerAddress":"PLOT NO.5222, PHASE- IV,"},{"dataSource":"API Import","activeIngredients":"","year":"2024","qtr":"Q1","strtotime":1710441000,"product":"(FOC) PAROXETINE HYDROCHLORIDE (PHARMACEUTICAL PRODUCTS)","address":"SITE NO.1,BOMMASANDRA INDL.AREA,IV","city":"ANEKAL TALUK,BANGALORE","supplier":"APOTEX INC","supplierCountry":"CANADA","foreign_port":"TORONTO","customer":"APOTEX INC","customerCountry":"INDIA","quantity":"0.27","actualQuantity":"274.6","unit":"GMS","unitRateFc":"3","totalValueFC":"831","currency":"USD","unitRateINR":"251.3","date":"15-Mar-2024","totalValueINR":"69009.99","totalValueInUsd":"831","indian_port":"Bangalore Air","hs_no":"29349990","bill_no":"2585804","productDescription":"API","marketType":"REGULATED MARKET","country":"CANADA","selfForZScoreResived":"Pharma Grade","supplierPort":"TORONTO","supplierAddress":"34,SPALDING DRIVEBRANTFORD ONTARIO N3T 6B8SDNF CA","customerAddress":"SITE NO.1,BOMMASANDRA INDL.AREA,IV"},{"dataSource":"API Import","activeIngredients":"","year":"2024","qtr":"Q3","strtotime":1721586600,"product":"PAROXETINE HYDROCHLORIDE HEMIHYDRATE","address":"PLOT NO. 1A, SECTOR 16A,","city":"NOIDA U.P.","supplier":"ZHEJIANG HUAHAI PHARMACEUTICAL","supplierCountry":"CHINA","foreign_port":"SHANGHAI","customer":"JUBILANT GENERICS","customerCountry":"INDIA","quantity":"50.00","actualQuantity":"50","unit":"KGS","unitRateFc":"660","totalValueFC":"33599","currency":"USD","unitRateINR":"55803","date":"22-Jul-2024","totalValueINR":"2790150","totalValueInUsd":"33599","indian_port":"Delhi Air","hs_no":"29349990","bill_no":"4630451","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"SHANGHAI","supplierAddress":"XUNQIAO.LINHAIZHEJIANG 317024,, CHINA","customerAddress":"PLOT NO. 1A, SECTOR 16A,"}]
18-Jan-2021
23-Aug-2024
KGS
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Average Price (USD/KGS)

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Total Value (USD)

Quantity (KGS) & Unit rate (USD/KGS) over time

API Imports and Exports

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